As the summer sun rises high, everyone anticipates relief from seasonal sniffles. Yet increasingly, more and more people are falling ill with cold-like symptoms in warmer weather too. The twist? A new Covid strain—NB.1.8.1—is spreading low-key around the world, from Asia to America and the UK. The challenge is distinguishing between a run-of-the-mill summer cold and a COVID-19 infection.

Typically, respiratory illnesses peak in the colder months when more time is spent indoors and dry air allows viruses to be more easily susceptible. Summer, however, is not virus-free. Parainfluenza virus Type 3, enteroviruses, adenoviruses, and even rhinoviruses are still causing issues in warmer climates. Social events, travel, lack of rest, more alcohol consumption, and air conditioning all set the stage for infections.

Complicating the situation further this summer is the discovery of the NB.1.8.1 Covid strain. Although the World Health Organization (WHO) only recently started monitoring it, scientists are closely monitoring its trajectory and possible implications.

Initially discovered in China in January 2025, NB.1.8.1 has a number of mutations that have caught the eye of the world's health authorities. The good news? There is as yet no evidence to suggest it results in more serious disease. In fact, U.K. Health Security Agency statistics indicate a modest rise in COVID cases with 5.2% of patients positive—up from 4.5% last week.

To date, just 13 of the confirmed NB.1.8.1 cases in the U.K. have been sequenced, the majority of them in April and May. While uncommon at present, the fact that it shares features with other variants of Covid means caution is still crucial.

Summer Colds vs. NB.1.8.1 Covid: Why It's Hard to Tell the Difference?

Both summer colds and Covid-19 have common symptoms such as:

• Sore throat
• Nasal congestion or runny nose
• Fatigue
• Muscle aches
• Nausea or vomiting

However some symptoms will tend more towards Covid, such as:

• Fever or chills
• Shortness of breath
• Loss of taste or smell, new
• Gastrointestinal illness such as diarrhea

There is no guaranteed way to tell the difference between a summer cold and Covid without doing a diagnostic test. Mild COVID-19 symptoms are easily mistaken for other seasonal viruses.

What Exactly Is a Summer Cold?

Colds—seasonal or not—are viral illnesses that are transmitted by respiratory droplets, fomites (contaminated surfaces), or close proximity. The main culprits are rhinoviruses, especially during the winter months, but the warmer months experience an upsurge in viruses such as parainfluenza, enteroviruses (coxsackie and echovirus), and adenoviruses.

Summer activities—weddings, concerts, holidays—promote intimate contact between groups, and typical summer behaviors such as drinking, bad sleep, and poor diet compromise immune systems. Air conditioning units, by dehydrating nasal passages, also impair the body's resistance to viral invaders.

The Centers for Disease Control and Prevention (CDC) urges anyone with cold-like symptoms to monitor closely. Covid-19 symptoms can appear two to 14 days after exposure and vary from mild to severe. The virus can be contagious two days before symptoms emerge and up to 10 days—or more—in immunocompromised individuals.

If you’re feeling unwell, the best course of action is to:

• Stay home and rest
• Wear a mask if around others
• Get a COVID-19 test
• Talk to your healthcare provider

Timely treatment, such as antiviral therapy with Paxlovid, is ensured through accurate diagnosis, particularly among high-risk individuals.

Can Vaccines Protect Against NB.1.8.1?

While a newer vaccine aimed at fall variants is being developed, the existing vaccines remain protective against NB.1.8.1. The variant is of the "drifter" type from the Omicron lineage, which indicates earlier immunity could still help prevent severe disease.

Dr. Aaron Chen, a Johns Hopkins University virologist, observes, "Although mutation is unavoidable, current vaccines remain effective in preventing hospitalization and complications from new variants, such as NB.1.8.1."

How To Avoid Getting Sick This Summer?

There's no surefire way to completely avoid viruses, unfortunately. But here are some practical tips from experts:

• Drink plenty of water and moderate your booze
• Consume a nutrient-dense diet full of fruits, vegetables, and lean proteins
• Get good rest and reduce stress levels
• Clean those surfaces that are touched most often such as airplane trays, phones, and exercise equipment
• Supplement with vitamin D if one is deficient since it enhances immune function
• Wash hands frequently, do not share food or beverages, and wear a mask in public indoor areas

What If You Do Get Sick?

Most people recover from summer colds and mild Covid-19 at home. Supportive care—hydration, over-the-counter pain relief, and rest—is typically enough. Nasal decongestants and lozenges can ease symptoms, while more serious or persistent cases warrant medical consultation.

If you suspect Covid, getting tested is crucial—not only for your health but for the wellbeing of those around you.

Hepatitis is not nicknamed the “silent killer” for nothing. What makes it so dangerous is how symptomless it often is for years. It can quietly chip away at your liver without so much as a warning sign. By the time symptoms like fatigue, jaundice, or abdominal pain appear, significant harm may already have occurred. This eerie silence allows hepatitis to go undetected until it has done some serious damage.

Just Because You Cannot Hear It Does Not Mean It Is Not There

Dr Vikram Vora says that hepatitis is a major global health threat, killing over a million people each year. What is even more tragic? These deaths are, in his words, “largely preventable”. The villains in this tale, cirrhosis and liver cancer, often stem from untreated or undiagnosed hepatitis infections.

Prevention Helps

“The cornerstone of hepatitis control is prevention,” Dr Vora says. And the good news is that you have got some powerful tools at your disposal. “Vaccination, especially against Hepatitis B, is one of the most effective tools available,” he says, adding that infants, healthcare workers, and at-risk adults should be prioritised. Other prevention measures include safe injection practices, steering clear of needle-sharing, and ensuring blood transfusions are properly screened.

But danger often hides in plain sight. Dr Vora warns that something as seemingly harmless as getting a tattoo or piercing can carry lifelong consequences if done in an unregulated facility. His advice? “Choosing safe, licensed establishments is non-negotiable.”

Why You Should Not Wait for Symptoms

If there is one thing hepatitis is good at, it is staying undetected. That is why Dr Vora champions regular screening, especially if you have had medical procedures, blood transfusions, or close contact with someone infected. “Early diagnosis is a game-changer,” he says. Caught in time, hepatitis can be treated effectively, helping patients avoid severe liver disease or even make a full recovery.

A Diagnosis Is Not a Death Sentence

Dr Vora offers hope to those living with hepatitis. “With proper medical care and lifestyle adjustments, a healthy life is achievable,” he insists. That means ditching alcohol, sticking to a balanced diet, maintaining a healthy weight, and closely following medical advice. He also points out the mental and emotional toll a diagnosis can take. “Support from family, community, and healthcare providers can make a significant difference in outcomes.”

The Stigma That Silences

Perhaps just as damaging as the virus itself is the social stigma surrounding it. Dr Vora identifies this as a major hurdle in tackling hepatitis. “Fear of social exclusion keeps many from seeking help,” he explains. And that delay can cost lives. According to him, building a culture of empathy and understanding is critical. He encourages public health campaigns, education, and open dialogue as essential tools to dismantle this stigma.

Make Noise Against the Silence

In Dr Vora’s words, “Hepatitis may be silent, but it is not invincible.” Every little step, whether it is getting vaccinated, getting screened, or just talking about it, can chip away at the power hepatitis holds. Silence has cost too many lives already. Awareness can save them.

If that viral video of a Gurugram woman being pounded by a pet husky dog for no reason scares you to death, you are not alone. Indian streets are turning into nightmares for people due to increasing dog bites. Reportedly, there is a sharp rise in dog bite cases, with over 3.7 million incidents and 54 confirmed human rabies deaths reported in 2024. The figures, compiled by the National Centre for Disease Control (NCDC) under the National Rabies Control Programme, show the grim situation and growing public health challenge.

Deaths caused by rabies also witnessed a sharp increase during the period. And this trend threatens India’s target of eliminating rabies by 2030 under its National Rabies Control Programme. While the woman was recently attacked by a pet Husky dog during a morning walk on the Gulf Course Road, no one is safe, especially the kids and the elderly.

The stray dog menace is taking over the streets, and here is everything you need to know about rabies and how it spreads.

If you think rabies only spreads through a bite, think again. While dog bites are the most well-known cause of Rabies infection, the virus can also be transmitted through something as simple as a lick, especially if it comes into contact with broken skin, the eyes, nose, or mouth. This is not a rare exception; it is a recognised mode of transmission and one that can be fatal if ignored.

Not to spook you into avoiding every friendly tail-wagger in your lane, but it is time we stopped thinking of rabies as a “bite-only” infection. This deadly disease does not wait around for dog attacks. It can infect you quietly, undetected, and by the time symptoms show up, it is usually too late.

The Lick That Could Kill

Rabies is caused by a virus present in the saliva of infected animals. While bites are the most common way this virus enters the human body, they are not the only route. If an infected animal licks a cut, scrape, broken skin, or even your eyes, nose, or mouth, the virus can get in. That means even cuddly encounters with pets, if they are unvaccinated and infected, can be risky.

The scary part is that you do not always feel or notice the moment it happens. A small scratch from a street pup, or a wet doggy kiss on a chapped lip, may not register as dangerous. But if the animal is rabid, that is all the opportunity the virus needs.

Do Not Wait for Symptoms

Rabies is one of the few diseases with a 99.9 per cent fatality rate once symptoms appear. It starts off with flu-like signs: fever, fatigue, and maybe a horrendous headache. But things quickly escalate. You can suffer hallucinations, paralysis, seizures, hydrophobia and eventually coma.

Once the virus reaches your nervous system, modern medicine throws up its hands. There is no cure.

But rabies is 100 per cent preventable if treated in time.

What You Should Do Immediately

The moment you realise you have been licked, scratched, or bitten by an animal, especially one that is unvaccinated or unknown, drop everything and:

1. Wash the wound thoroughly with soap and running water for at least 15 minutes. This one step could significantly reduce your risk.

2. Apply antiseptic like iodine or alcohol.

3. Rush to a doctor; do not wait for swelling, itching or any other sign. You will need a post-exposure prophylaxis (PEP) shot, which includes a course of anti-rabies vaccines and sometimes immunoglobulin injections.

4. Keep a record of the animal if possible. If it is someone’s pet, you will want to confirm their vaccination history.

Dogs Are Not the Only Issue

While dogs are responsible for 95 per cent of rabies cases in India, they are not the only suspects. Cats, monkeys, bats, and even cows can carry the virus. Basically, any warm-blooded mammal is fair game. So if a cheeky monkey in a temple town swipes at you, or a bat dive-bombs your balcony and leaves a scratch, take it seriously.

You do not need a bite to get rabies. You just need a moment of bad luck and a droplet of infected saliva. But that does not mean we need to start treating every dog badly. It just means we need to be alert, informed, and quick to act.

'Medical Memoir' is a Health & Me series that delves into some of the most intriguing medical histories and unveils how medical innovations have evolved over time. Here, we trace the early stages of all things health, whether a vaccine, a treatment, a pill, or a cure.

You think this is a history lesson, but let’s do some math. What does Hepato‑ (“liver”) + ‑tropic (“infecting and replicating in the liver”) + DNA (HBV genome) equal? That spells hepatitis—a mystery virus long misunderstood, long before COVID dominated headlines.

This isn’t just linguistic trivia. It’s a journey through centuries of medical detective work, flawless missteps, and public health triumphs—all centered on one family of viruses that quietly reshaped liver medicine and modern virology.

Early Clues in Jaundice and Contagion

Hepatitis didn’t appear on a microscope slide in the 20th century—it was described millennia earlier. Ancient Greek physician Hippocrates observed jaundice as a distinctive yellowing of skin and eyes. By the 8th century, medical writers suspected jaundice was contagious, hinting at an infectious agent at work in the liver.

Between the 17th and 19th centuries, dramatic epidemics of jaundice spread through soldiers and navies. Shared living quarters, contaminated water, and later, mass inoculation campaigns and reused syringes during wars linked the outbreaks to bloodborne transmission. By 1885, physicians recognized that transfused blood could carry the mysterious jaundice-causing pathogen. And when measles and yellow fever vaccinations during World War II triggered outbreaks, it became clear: an unseen virus replicated in bloodstreams—and in the liver.

This early history underscored two enduring facts: hepatitis came in different forms, and it was stealthy, delayed, and extremely hard to track.

Naming a Virus in Two Parts

By mid-century, researchers sought to categorize the illness. In 1947, MacCallum introduced a dual classification that would shape all future work: Viral Hepatitis A (infectious, via contaminated food or water) and Viral Hepatitis B (serum-borne, via blood and body fluids). That breakthrough turned a single symptom—jaundice—into a family of diseases with different transmission pathways and prevention needs.

Modern hepatitis history truly began in the 1960s. In 1963, Baruch Blumberg identified the Australia antigen (HBsAg) in Aboriginal Australians and hemophiliac blood donors using immunologic screening. It was the first direct marker for Hepatitis B virus, revealing a virus that lay dormant in carriers, often for decades.

The story advanced quickly:

1970 – The Dane particle (complete HBV virus) was visualized, proving the viral identity.

Early 1970s – Researchers described the HBeAg, another disease marker.

1973 – Hepatitis A virus (HAV) was isolated, confirming two different viruses.

Suddenly, hepatitis was no longer a vague syndrome—it was a family of identifiable pathogens. That scientific clarity laid the foundation for prevention and treatment.

Finding a Cure Before HCV With Plasma‑Derived Vaccines

The early HBV vaccine story is unique. Developed from human plasma, it became the first vaccine not produced via tissue culture, licensed under the name Heptavax in 1981. That innovation paved the way for recombinant vaccines, but the achievement stood alone for a time. Taiwan’s national HBV vaccination program, launched in 1984, offered powerful proof of impact:

HBV carrier rate dropped from 9.1% to 2.7% among children.

Rates of pediatric hepatocellular carcinoma plummeted from 27% to 17%.

Effectively, this was the world’s first anti-cancer vaccine. The vaccine didn’t just stop liver infection—it significantly reduced childhood liver cancers in real populations.

The C Virus and the Molecular Breakthrough of 1989

In 1989, the hepatitis world shifted again. A team led by Choo, Kuo, and Houghton unveiled Hepatitis C virus (HCV) using molecular cloning techniques—no tissue culture, electron microscope, or serology required. It was the first virus discovered solely via genetic methods.

That breakthrough explained persistent post‑transfusion hepatitis cases, intravenous drug-user outbreaks, and a heavy burden of global liver disease. It was the start of a new era: molecular virology, where pathogens were identified by their RNA alone.

Soon came discoveries of HDV (delta virus), HEV, and GBV‑C/HGV (hepatitis G virus). This growing catalog of viral types clarified why hepatitis symptoms varied and why prevention needed multiple strategies.

From Unproven Remedies to Modern Antivirals

Mid-20th-century treatment was often worse than the disease: prolonged bed rest and steroids were used for acute hepatitis until controlled trials in the ’60s showed they caused harm. In chronic cases, steroids lingered until the 1980s, when researchers clearly proved they worsened outcomes.

By the early 1990s, interferon alpha offered the first real antiviral option. In 1998, lamivudine appeared, followed by more potent nucleoside analogues in the 2000s. For chronic HBV, these drugs allowed suppression of the virus with daily pills—a huge leap in quality of life. HCV treatment advanced spectacularly:

• Pegylated interferon plus ribavirin drove cure rates into the 40–60% range.
• Protease inhibitors like boceprevir and telaprevir added another 30%+ success.
• By the 2010s, direct-acting antivirals (DAAs) offered cure rates above 85–90% within a few months, without interferon.
• Chronic HCV became the only curable chronic viral infection—a feat comparable in scope to eradication campaigns for smallpox or polio.

Globally today, over 300 million people live with chronic HBV or HCV and over 1.3 million die annually from liver cirrhosis, cancer, or hepatitis complications.

In response, the World Health Organization’s 2022–2030 strategy aims to reduce new infections by 90% and deaths by 65%. It underscores the urgent need for universal vaccinations, access to testing, and treatment—especially in low-income settings across Asia and Africa, without coordinated action, projections warn:

• 9.5 million new infections,
• 2.1 million liver cancer cases,
• 2.8 million deaths by 2030.

Yet history offers reason for optimism: the leaps science made over just a few decades show what’s possible—with political will, investment, and global partnerships.

HBV opened new pathology understanding by linking to polyarteritis nodosa. HCV revealed risks beyond the liver: cryoglobulinemia, non-Hodgkin lymphoma, metabolic disorders like Type II diabetes, and altered lipid profiles. Hepatitis viruses reshaped fields ranging from rheumatology to oncology.

The discoveries required collaboration—and some competition—across labs and continents. The story of hepatitis is one of scientific rivalry, teamwork, serendipity, and intellectual persistence. That blend of curiosity and rigor transformed a family of mysterious illnesses into preventable and treatable diseases.

Long before COVID captured global focus—before masks, PCR tests, and lockdowns—hepatitis was a mystery virus that haunted generations. It caused jaundice, silent liver failure, and cancer. It eluded diagnosis, lacked vaccines, and spread undetected.

Yet through a combination of linguistic insight, molecular science, epidemiology, and public health action, the world unraveled its secrets—one virus at a time.

Today, hepatitis is no longer an invisible threat. It's a medical triumph—because when science is patient, and public health persistent, even the most elusive pathogens can be conquered. The story isn't over, but it's a testament to what decades of dedication and discovery can achieve.