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Having a morning coffee to start your day, or a midday coffee to keep your day going, is the best feeling. It energizes you and helps you get through the day. But have you ever noticed how you are left with sour notes in your mouth and a foul smell. Does that mean you have bad hygiene or is it an indicator that you should not be having coffee?
The answer is neither, it is due to some compounds in the coffee. You may have noticed that this is the case with many other strong-tasting foods like garlic, onions, boiled chicken and other dairy products. The Mayo Clinic explains that food particles breakdown around your teeth leads to more bacteria and causes a bad smell. These foods enter the blood stream after you ingest, get carried to your lungs and affect one’s breath.
While it may seem like a minor issue, it can be noticeable to others and make us feel self-conscious. So, it's important to understand why coffee causes bad breath and what we can do to get rid of it.
When coffee beans are roasted, they release certain chemicals that contain sulfur. According to MedlinePlus these sulfur compounds are a big reason why coffee makes your breath smell bad. Think of it like the smell of rotten eggs, but much milder. Coffee is also acidic, which means it can change the pH balance in your mouth, making it easier for bacteria to grow.
When bacteria grow, they release even more smelly sulfur compounds. On top of that, coffee can dry out your mouth. Caffeine acts a little like a dehydrating agent, and coffee also contains tannins, which stop your mouth from making enough saliva. Saliva helps wash away bacteria and food particles, so when your mouth is dry, these things stay around and cause bad breath.
The best way to avoid coffee breath is to make some smart choices about how you drink your coffee. Experts suggest drinking black coffee, without any milk or sugar, is better for your breath. Milk and sugar can feed the bacteria in your mouth, making the smell worse. If you must add flavor, try stirring your coffee with a cinnamon stick or a vanilla bean. These natural flavorings can add a nice taste without causing bad breath. If you need a sweetener, try using sugar substitutes instead of regular sugar. If you have to use dairy, whole milk or half and half are better than skim milk, as skim milk contains more sugar.
If you're willing to try something other than coffee, there are some good alternatives that can still give you a caffeine boost. High-caffeine black tea or chai tea can provide a similar pick-me-up without the strong coffee smell.
Whether you drink coffee or not, good oral hygiene is essential for fresh breath. The sulfur compounds, acidity, and tannins in coffee can all contribute to bad breath. So, it's important to brush your teeth twice a day, floss regularly, and use mouthwash. If you drink coffee, you might need to pay extra attention to your oral hygiene
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A retracted eardrum, also called tympanic membrane atelectasis, is a condition where the eardrum gets pulled inward toward the middle ear. Normally, the eardrum (or tympanic membrane) acts as a boundary between the outer and middle ear, transmitting sound vibrations to tiny bones that help us hear. But when it collapses inward, that delicate process can be disrupted.
The condition is often silent at first, meaning people may not realize they have it. But in some cases, it can escalate, causing discomfort or even permanent hearing damage.
In most cases, a retracted eardrum doesn’t cause any noticeable symptoms. But when the retraction becomes severe enough to affect structures inside the ear, individuals may experience:
If left untreated, a chronic retracted eardrum can lead to permanent hearing loss.
The most common cause of a retracted eardrum is Eustachian tube dysfunction. These narrow tubes connect the middle ear to the back of the nose and help regulate ear pressure.
When they don’t work properly, pressure inside the ear drops, effectively pulling the eardrum inward.
Some common triggers include:
Upper respiratory infections, such as the common cold
Diagnosis typically begins with a discussion about symptoms and any recent infections. A doctor will then use an otoscope, a tool with a light, to look into the ear canal. This allows them to visually confirm if the eardrum is abnormally positioned or collapsed.
Not all retracted eardrums require immediate treatment. In mild cases, doctors often recommend a “watch and wait” approach, as pressure may normalize naturally over a few months.
For more advanced cases, several interventions are possible:
Decongestants or nasal steroids: These can improve airflow in the ear and relieve pressure.
The Valsalva maneuver: This self-administered technique involves closing your mouth, pinching your nose, and gently blowing as if trying to pop your ears. It should be performed under medical guidance.
If the condition begins to affect hearing or causes persistent pain, surgical options may be considered.
Two common surgical treatments are:
Tube Insertion (Myringotomy):
Often used in children with recurring ear infections, this procedure involves placing small tubes into the eardrum to help ventilate the middle ear.
Tympanoplasty:
In more severe cases, part of the damaged eardrum may be removed and replaced with cartilage from the outer ear. This stiffens the eardrum, preventing future collapses.
The outlook largely depends on the severity. Minor retractions usually resolve without intervention and don’t cause long-term damage. However, more serious cases, especially those that persist or press against ear bones, may result in hearing loss and need medical or surgical correction.
Experts recommend seeking medical attention if you notice ear discomfort, hearing changes, or frequent infections. Early diagnosis can prevent long-term issues and protect one of your most important senses, your hearing.
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In a surprising discovery that bridges the gap between creepy crawlies and cutting-edge neuroscience, researchers at Virginia Tech have identified unique compounds in millipede secretions that could pave the way for future treatments for pain and neurological diseases like Parkinson’s, depression, and schizophrenia.
Led by chemist Emily Meyers, the research team uncovered naturally occurring alkaloids in the defensive secretions of the Andrognathus corticarius, a species known colloquially as the Hokie millipede. The millipede, which lives under decomposing leaves and branches on the university’s Blacksburg campus, produces a chemical cocktail that not only deters predators but may influence neuroreceptors in the brain.
“These compounds are quite complex, so they’re going to take some time to synthesize in the lab,” said Meyers, who specializes in studying underexplored ecological sources for potential new drugs.
The compounds, dubbed andrognathanols and andrognathines by Meyers’ team, belong to a class of complex alkaloids. They were discovered after researchers collected several millipedes from wooded areas on campus and analyzed the contents of their defensive glands using a suite of chemical tools.
The results were striking: some of these secretions had a disorienting effect on ants, one of the millipede’s presumed predators. But that’s not all, several of the compounds were found to interact with a neuroreceptor known as Sigma-1. This receptor has been linked to multiple brain disorders, including schizophrenia, depression, Lou Gehrig’s disease (ALS), and Parkinson’s disease.
In addition to warding off predators, the researchers discovered that the compounds may also serve a social function, possibly helping millipedes signal their location to family members in leaf-littered environments.
This study, recently published in the Journal of the American Chemical Society, is not Meyers’ first foray into studying arthropod chemistry. She has been collaborating with entomologist Paul Marek, and together, they have previously suggested that the family of alkaloids found in millipede secretions could have significant therapeutic potential.
“Millipedes have been around for hundreds of millions of years. They’ve developed these intricate chemical defense systems, and we’re only beginning to understand their value,” said Meyers.
While the compounds show promise, the next hurdle is a familiar one in drug discovery: scalability. The compounds exist in trace amounts in the wild, and researchers need larger quantities for in-depth testing and potential pharmaceutical development.
The team is now exploring partnerships with laboratories that can synthesize the compounds in bulk, which would allow for further testing on their biological activity and medicinal properties. Meyers emphasized that while the research is still in its early stages, the potential applications are broad, from pain management to novel treatments for complex neurological conditions.
“Nature has always been a wellspring of inspiration for medicine,” said Meyers. “And sometimes, the most powerful solutions come from the smallest and most unexpected creatures, like a tiny millipede under a log.”
With this groundbreaking discovery, scientists are reminded once again that the natural world may hold secrets that, once unlocked, could transform human health in unimaginable ways.
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Cancers are unpredictable and can be either containable or completely wreck your health. To make cancer treatments and research better, healthcare professionals use stages and characteristics, making it easier for us to categorize and identify how far along the disease is. However, recent research has been questioning our stance on this particular cancer, saying it could be worse than what we had believed.
A recent study has found that a specific type of prostate tumor, called Grade Group one (GG1), might not be as harmless as once believed. While many doctors consider these tumors to be at low risk of spreading and recommend monitoring them, the new research suggests the true risk might be higher. In fact, the study indicates that up to 30% of these cases could be more serious than doctors think, and a closer look could save lives.
For years, men diagnosed with a GG1 tumor were often told to skip immediate treatment and instead undergo "active surveillance." This means they were regularly checked with blood tests and follow-up biopsies to see if the tumor was growing.
However, researchers point out a key problem: a single biopsy might miss more aggressive cancer cells located in a different part of the prostate. This could lead to a patient being undertreated, which could have serious consequences later on if the cancer grows. The authors believe that relying on just one biopsy to decide on treatment is a flawed approach.
According to a 2022 review published in the Urological Research Society, the lowest grade of prostate cancer, called ISUP Grade Group 1 (GG1), grows very slowly. Because it is so harmless, some experts have suggested that we should stop calling it "cancer" at all. This has been done before for similar low-risk tumors in the bladder and thyroid.
The review summarized that even though GG1 tumors have some characteristics of cancer, their behavior is much more like a harmless, non-cancerous growth. The authors suggest that renaming GG1 prostate cancer could have several benefits:
Challenging the view of previous studies, researchers sought to get a clearer understanding. To get a more accurate picture, the research team looked at data from over 300,000 men. Among those who were initially diagnosed with GG1 tumors, the team used additional factors like PSA levels and tumor size, which can be better indicators of risk.
By combining all this information, they found that a significant number of men—more than 18,000—were actually at higher risk and should have received more aggressive treatment like radiation or surgery. This data strongly suggests that as many as 30% of GG1 diagnoses might be underestimated, leaving many men without the full treatment they need.
The authors of the study are urging doctors to reconsider how they evaluate GG1 tumors. They emphasize that a "low grade" diagnosis from a biopsy isn't the same as a guaranteed "low risk" for the patient. They believe it's a doctor's responsibility to use all available data to accurately assess a man's individual risk. The goal is to ensure that those who need treatment get it, while still safely recommending active surveillance for the men who are truly at low risk.
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