Ananda Lewis, celebrated TV host known for her authenticity and warmth, passed away at 52 after a long and brave battle with breast cancer. Once a beloved face on BET’s Teen Summit and MTV’s Total Request Live, she used her platform to engage young audiences with pressing social issues and celebrity interviews that left a mark.

A Powerful Presence on Screen

Lewis first came into the spotlight through BET’s Teen Summit, where she led candid conversations on challenges facing Black youth. Her interviews with figures like Kobe Bryant, Tupac Shakur, and Hillary Clinton earned her critical acclaim, including an NAACP Image Award.

In 1997, she joined MTV, becoming a key voice on MTV Live, Hot Zone, and TRL. Though her move sparked criticism, she remained unapologetic. “Growth is necessary,” she told the Associated Press. “You’ve got to get out there and live your life.”

She later launched The Ananda Lewis Show in 2001, a daytime talk show aimed at real conversations, even if it followed a familiar format. The show lasted one season, but Lewis remained a powerful presence, later joining The Insider as a correspondent.

Breast Cancer Journey That She Shared With The World

In 2020, Lewis revealed that she had been living with stage 3 breast cancer since 2018—later advancing to stage 4. Her diagnosis came after years of ignoring persistent inflammation linked to mastitis from breastfeeding. A lump discovered in her right breast led to a biopsy, confirming it was invasive carcinoma.

Instead of following a conventional medical path, Lewis chose alternative treatments. “I wanted to understand why my body created cancer and how to change the terrain,” she explained. She focused on diet changes, stress management, detoxing, and holistic therapies like high-dose vitamin C, acupuncture, and cryoablation.

A Different Kind of Battle

Lewis moved to Arizona in 2020 for 16 weeks of intensive integrative treatment. Her cancer reduced from stage 3 to 2 and was no longer in her lymph nodes. But the journey wasn’t without hardship—financial struggles and insurance lapses made consistent treatment difficult.

In early 2023, a scan showed the tumor had grown slightly, but it had not spread. She underwent electrical ablation in Mexico, which unfortunately didn’t succeed. By October 2023, a PET scan confirmed the cancer had advanced to stage 4. Still, Lewis continued treatment and saw significant improvement by January 2024.

Lewis was candid about her journey, using her story to raise awareness about early detection and holistic health. “Stage 4 doesn’t mean the end,” she said. “It means I have to do more.”

Her message to women was clear: be proactive. Prioritize mammograms, manage stress, sleep well, and adopt a clean lifestyle. “If I had known what I know now ten years ago, perhaps I wouldn’t have ended up here,” she said.

“I want to look back and say—I did that exactly how I wanted to,” Lewis reflected. Her legacy is one of resilience, wisdom, and an enduring belief in the right to choose one’s own path—even through cancer.

If that viral video of a Gurugram woman being pounded by a pet husky dog for no reason scares you to death, you are not alone. Indian streets are turning into nightmares for people due to increasing dog bites. Reportedly, there is a sharp rise in dog bite cases, with over 3.7 million incidents and 54 confirmed human rabies deaths reported in 2024. The figures, compiled by the National Centre for Disease Control (NCDC) under the National Rabies Control Programme, show the grim situation and growing public health challenge.

Deaths caused by rabies also witnessed a sharp increase during the period. And this trend threatens India’s target of eliminating rabies by 2030 under its National Rabies Control Programme. While the woman was recently attacked by a pet Husky dog during a morning walk on the Gulf Course Road, no one is safe, especially the kids and the elderly.

The stray dog menace is taking over the streets, and here is everything you need to know about rabies and how it spreads.

If you think rabies only spreads through a bite, think again. While dog bites are the most well-known cause of Rabies infection, the virus can also be transmitted through something as simple as a lick, especially if it comes into contact with broken skin, the eyes, nose, or mouth. This is not a rare exception; it is a recognised mode of transmission and one that can be fatal if ignored.

Not to spook you into avoiding every friendly tail-wagger in your lane, but it is time we stopped thinking of rabies as a “bite-only” infection. This deadly disease does not wait around for dog attacks. It can infect you quietly, undetected, and by the time symptoms show up, it is usually too late.

The Lick That Could Kill

Rabies is caused by a virus present in the saliva of infected animals. While bites are the most common way this virus enters the human body, they are not the only route. If an infected animal licks a cut, scrape, broken skin, or even your eyes, nose, or mouth, the virus can get in. That means even cuddly encounters with pets, if they are unvaccinated and infected, can be risky.

The scary part is that you do not always feel or notice the moment it happens. A small scratch from a street pup, or a wet doggy kiss on a chapped lip, may not register as dangerous. But if the animal is rabid, that is all the opportunity the virus needs.

Do Not Wait for Symptoms

Rabies is one of the few diseases with a 99.9 per cent fatality rate once symptoms appear. It starts off with flu-like signs: fever, fatigue, and maybe a horrendous headache. But things quickly escalate. You can suffer hallucinations, paralysis, seizures, hydrophobia and eventually coma.

Once the virus reaches your nervous system, modern medicine throws up its hands. There is no cure.

But rabies is 100 per cent preventable if treated in time.

What You Should Do Immediately

The moment you realise you have been licked, scratched, or bitten by an animal, especially one that is unvaccinated or unknown, drop everything and:

1. Wash the wound thoroughly with soap and running water for at least 15 minutes. This one step could significantly reduce your risk.

2. Apply antiseptic like iodine or alcohol.

3. Rush to a doctor; do not wait for swelling, itching or any other sign. You will need a post-exposure prophylaxis (PEP) shot, which includes a course of anti-rabies vaccines and sometimes immunoglobulin injections.

4. Keep a record of the animal if possible. If it is someone’s pet, you will want to confirm their vaccination history.

Dogs Are Not the Only Issue

While dogs are responsible for 95 per cent of rabies cases in India, they are not the only suspects. Cats, monkeys, bats, and even cows can carry the virus. Basically, any warm-blooded mammal is fair game. So if a cheeky monkey in a temple town swipes at you, or a bat dive-bombs your balcony and leaves a scratch, take it seriously.

You do not need a bite to get rabies. You just need a moment of bad luck and a droplet of infected saliva. But that does not mean we need to start treating every dog badly. It just means we need to be alert, informed, and quick to act.

'Medical Memoir' is a Health & Me series that delves into some of the most intriguing medical histories and unveils how medical innovations have evolved over time. Here, we trace the early stages of all things health, whether a vaccine, a treatment, a pill, or a cure.

You think this is a history lesson, but let’s do some math. What does Hepato‑ (“liver”) + ‑tropic (“infecting and replicating in the liver”) + DNA (HBV genome) equal? That spells hepatitis—a mystery virus long misunderstood, long before COVID dominated headlines.

This isn’t just linguistic trivia. It’s a journey through centuries of medical detective work, flawless missteps, and public health triumphs—all centered on one family of viruses that quietly reshaped liver medicine and modern virology.

Early Clues in Jaundice and Contagion

Hepatitis didn’t appear on a microscope slide in the 20th century—it was described millennia earlier. Ancient Greek physician Hippocrates observed jaundice as a distinctive yellowing of skin and eyes. By the 8th century, medical writers suspected jaundice was contagious, hinting at an infectious agent at work in the liver.

Between the 17th and 19th centuries, dramatic epidemics of jaundice spread through soldiers and navies. Shared living quarters, contaminated water, and later, mass inoculation campaigns and reused syringes during wars linked the outbreaks to bloodborne transmission. By 1885, physicians recognized that transfused blood could carry the mysterious jaundice-causing pathogen. And when measles and yellow fever vaccinations during World War II triggered outbreaks, it became clear: an unseen virus replicated in bloodstreams—and in the liver.

This early history underscored two enduring facts: hepatitis came in different forms, and it was stealthy, delayed, and extremely hard to track.

Naming a Virus in Two Parts

By mid-century, researchers sought to categorize the illness. In 1947, MacCallum introduced a dual classification that would shape all future work: Viral Hepatitis A (infectious, via contaminated food or water) and Viral Hepatitis B (serum-borne, via blood and body fluids). That breakthrough turned a single symptom—jaundice—into a family of diseases with different transmission pathways and prevention needs.

Modern hepatitis history truly began in the 1960s. In 1963, Baruch Blumberg identified the Australia antigen (HBsAg) in Aboriginal Australians and hemophiliac blood donors using immunologic screening. It was the first direct marker for Hepatitis B virus, revealing a virus that lay dormant in carriers, often for decades.

The story advanced quickly:

1970 – The Dane particle (complete HBV virus) was visualized, proving the viral identity.

Early 1970s – Researchers described the HBeAg, another disease marker.

1973 – Hepatitis A virus (HAV) was isolated, confirming two different viruses.

Suddenly, hepatitis was no longer a vague syndrome—it was a family of identifiable pathogens. That scientific clarity laid the foundation for prevention and treatment.

Finding a Cure Before HCV With Plasma‑Derived Vaccines

The early HBV vaccine story is unique. Developed from human plasma, it became the first vaccine not produced via tissue culture, licensed under the name Heptavax in 1981. That innovation paved the way for recombinant vaccines, but the achievement stood alone for a time. Taiwan’s national HBV vaccination program, launched in 1984, offered powerful proof of impact:

HBV carrier rate dropped from 9.1% to 2.7% among children.

Rates of pediatric hepatocellular carcinoma plummeted from 27% to 17%.

Effectively, this was the world’s first anti-cancer vaccine. The vaccine didn’t just stop liver infection—it significantly reduced childhood liver cancers in real populations.

The C Virus and the Molecular Breakthrough of 1989

In 1989, the hepatitis world shifted again. A team led by Choo, Kuo, and Houghton unveiled Hepatitis C virus (HCV) using molecular cloning techniques—no tissue culture, electron microscope, or serology required. It was the first virus discovered solely via genetic methods.

That breakthrough explained persistent post‑transfusion hepatitis cases, intravenous drug-user outbreaks, and a heavy burden of global liver disease. It was the start of a new era: molecular virology, where pathogens were identified by their RNA alone.

Soon came discoveries of HDV (delta virus), HEV, and GBV‑C/HGV (hepatitis G virus). This growing catalog of viral types clarified why hepatitis symptoms varied and why prevention needed multiple strategies.

From Unproven Remedies to Modern Antivirals

Mid-20th-century treatment was often worse than the disease: prolonged bed rest and steroids were used for acute hepatitis until controlled trials in the ’60s showed they caused harm. In chronic cases, steroids lingered until the 1980s, when researchers clearly proved they worsened outcomes.

By the early 1990s, interferon alpha offered the first real antiviral option. In 1998, lamivudine appeared, followed by more potent nucleoside analogues in the 2000s. For chronic HBV, these drugs allowed suppression of the virus with daily pills—a huge leap in quality of life. HCV treatment advanced spectacularly:

• Pegylated interferon plus ribavirin drove cure rates into the 40–60% range.
• Protease inhibitors like boceprevir and telaprevir added another 30%+ success.
• By the 2010s, direct-acting antivirals (DAAs) offered cure rates above 85–90% within a few months, without interferon.
• Chronic HCV became the only curable chronic viral infection—a feat comparable in scope to eradication campaigns for smallpox or polio.

Globally today, over 300 million people live with chronic HBV or HCV and over 1.3 million die annually from liver cirrhosis, cancer, or hepatitis complications.

In response, the World Health Organization’s 2022–2030 strategy aims to reduce new infections by 90% and deaths by 65%. It underscores the urgent need for universal vaccinations, access to testing, and treatment—especially in low-income settings across Asia and Africa, without coordinated action, projections warn:

• 9.5 million new infections,
• 2.1 million liver cancer cases,
• 2.8 million deaths by 2030.

Yet history offers reason for optimism: the leaps science made over just a few decades show what’s possible—with political will, investment, and global partnerships.

HBV opened new pathology understanding by linking to polyarteritis nodosa. HCV revealed risks beyond the liver: cryoglobulinemia, non-Hodgkin lymphoma, metabolic disorders like Type II diabetes, and altered lipid profiles. Hepatitis viruses reshaped fields ranging from rheumatology to oncology.

The discoveries required collaboration—and some competition—across labs and continents. The story of hepatitis is one of scientific rivalry, teamwork, serendipity, and intellectual persistence. That blend of curiosity and rigor transformed a family of mysterious illnesses into preventable and treatable diseases.

Long before COVID captured global focus—before masks, PCR tests, and lockdowns—hepatitis was a mystery virus that haunted generations. It caused jaundice, silent liver failure, and cancer. It eluded diagnosis, lacked vaccines, and spread undetected.

Yet through a combination of linguistic insight, molecular science, epidemiology, and public health action, the world unraveled its secrets—one virus at a time.

Today, hepatitis is no longer an invisible threat. It's a medical triumph—because when science is patient, and public health persistent, even the most elusive pathogens can be conquered. The story isn't over, but it's a testament to what decades of dedication and discovery can achieve.

First impressions can be deceiving, but they can also set the tone for your future interactions. A person who you perceive to be difficult will remain so in your eyes, until they actively prove otherwise. If that person keeps behaving as he is, even if their actions are not always unjustifiable, one finds ways to back their beliefs. Did you know this could also apply to babies, even before they are born?

A recent study done by researchers at University of Notre Dame suggests that the way medical staff talk during an ultrasound can really influence how new parents feel about their baby. For many people, the ultrasound is the very first time they get a glimpse of their child. If a technician blames a poor image on the baby being "uncooperative," this small comment can set a negative tone.

This kind of language can stick with parents, affecting how they see their child even before birth and possibly leading to less involved parenting later on. The study, published in Communications Psychology, highlights how these seemingly small word choices can have a lasting impact.

How Early Perceptions Affect Parenting?

The research found a clear connection between how mothers felt about their baby during pregnancy and how they viewed their toddler later on. Women who had a more positive view of their unborn child during the study reported fewer emotional or behavioral issues in their children at 18 months old.

On the other hand, mothers who had a more negative view of their unborn baby were more likely to report that their toddlers had trouble with things like attention, managing their emotions, and sleeping. This link was shown in an experiment where mothers who were told their baby was "uncooperative" were more likely to have a negative view of their child later.

Does A Child’s Behavior Affect Parents’ Bond?

Previous research has shown that a baby’s behavior can affect how the parents see them. According to the Frontiers in Public Health connection between how parents perceive their baby's behavior and the parents' own mental health and bonding with the baby. Researchers surveyed 73 mothers of infants between 3.5 weeks and 6 months old. The study found that when mothers reported their infants had frequent stomach or crying issues, they were also more likely to have:

• Bonding issues with their baby
• Higher levels of anxiety
• Higher levels of depression

The researchers believe that asking parents about their baby's behavior could be a simple way for healthcare professionals to identify mothers who need extra support and babies who may need additional help.

This study highlighted a new way for doctors and nurses to help new parents. Instead of only looking at risk factors like a parent's age, they could use simple questionnaires to ask about a parent's perception of their baby's behavior. This approach could make it easier to find families who need support, ensuring they get targeted help more quickly and efficiently.

This could lead to better relationships between parents and their babies and improve a child's long-term outcomes. The study also emphasizes the need for more research, particularly with larger, more diverse groups of people, to better understand these connections.

Importance of Mindful Communication

The findings of this study show just how important it is for healthcare providers to be careful with their words. Simple comments made during a critical moment like an ultrasound can have a lasting impact on parents.

The gap between patients and their healthcare professionals widen when communication lacks. This was shown in a 2020 survey published in the JAMA Open Network. The study found that more than 41% of women reported holding back from asking their clinicians questions during prenatal appointments. They reported several reasons for their reluctance like the clinician seemed rushed, they didn’t want to seem difficult, as well as preference to a different plan than what the clinicians wanted.

This careful communication could even play a role in a mother's risk for postpartum depression. Since the bond between a parent and child begins to form so early, a negative experience during an ultrasound could potentially affect this crucial relationship, influencing the well-being of both the parent and the child over time.