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Modifiable risk factors such as quitting smoking, exercising regularly, and eating a balanced diet can certainly help protect your heart. However, when it comes to heart disease, the role of family history also deserves serious attention. In other words, are cardiovascular heart diseases (CHD) hereditary? And if so, what can be done to lower your risk?
Genetics account for up to half of a person's risk for cardiovascular disease. While it is only one among many contributing factors, it can be a significant—and often early—clue to a person’s potential for heart-related issues. Having close relatives like a parent or sibling with heart disease can increase the chances of developing a heart condition. Shared environmental factors within families, such as dietary habits and levels of physical activity, also influence heart health.
Learning about family history can empower individuals to take charge of their healthcare. This knowledge allows potential health concerns to be identified before a serious event occurs and helps prevent disease progression. Identifying a genetic diagnosis within the family enables both patients and healthcare providers to stay current on recommended screenings and treatments.
Heart disease is often a combination of genetic and environmental influences. In certain cases, a gene may be passed down from one or both parents. If a parent carries a specific heart disease gene, there is a 50 per cent chance it could be inherited. Thus, parents can offer insight into potential future risks from a genetic standpoint.
Knowing family history helps clinicians assess whether a patient is at higher-than-average risk. It’s not just about anticipating what might happen—screenings and treatments based on genetic risk can offer more proactive management.
Several types of heart conditions can be inherited. These include coronary heart disease (blockages in arteries supplying the heart), congenital heart disease (a defect present at birth), cardiomyopathy (abnormal heart function without obvious cause), high cholesterol (which raises plaque risk), arrhythmias (irregular heart rhythms), and dilated aorta (an enlarged major blood vessel).
Although family history cannot be changed, modifiable risk factors can still be addressed. High cholesterol or blood pressure often shows up in families around the same age, but making lifestyle changes early can help. A low-fat, low-cholesterol, and low-sugar diet, along with at least 30 minutes of exercise five days a week, can help mitigate risk. Working with a cardiologist can help determine whether lifestyle changes or early medication are needed.
Collecting family history includes asking relatives about medications, diagnoses of high cholesterol or blood pressure, cardiac conditions, sudden or unexplained deaths, use of pacemakers or defibrillators, and whether anyone sees a cardiologist or has had heart surgery.
After gathering this information, a physician can recommend genetic testing if necessary. Certain patterns, like multiple relatives with early heart attacks or high cholesterol, may point to inherited conditions such as familial hypercholesterolemia. Even in the absence of family history, conditions like aortopathy may qualify someone for genetic testing.
Genetic testing typically starts with a three-generation family history and involves either a saliva or blood sample. Results may be positive, negative, or uncertain. Even a negative result doesn’t rule out a hereditary component, so follow-ups and testing for other family members may still be recommended.
Moreover, positive results may lead to cascade testing—where relatives are tested for the same condition—and can influence treatment strategies. Precision medicine and gene therapy now offer the possibility of correcting genetic defects at a molecular level, offering new hope for those at risk.
Credits: Canva
The 2025 flu season has turned out to be unlike any other in recent memory. This year, the flu season is marked by record-setting infections, multiple viral peaks, vaccine mismatches, and an overstretched healthcare system.
According to the Centers for Disease Control and Prevention (CDC), this year’s influenza activity is the most intense since the 2009 swine flu pandemic, with over 80 million estimated illnesses and rising. What’s driving this intensity, and what should the public know about prevention and symptom management?
Let’s break down what makes this flu season so severe and what it means for your health.
In most years, flu activity in the U.S. typically follows a predictable pattern, starting in October, peaking between December and February, and fading by April. But the 2024–2025 season has defied that rhythm.
Cases surged past the national baseline in December and then, unexpectedly, peaked again in February, a second wave that blindsided doctors and public health experts.
As of March 2025, the CDC had reported an estimated 37 million influenza infections, 480,000 hospitalizations, and 21,000 deaths. Hospitalizations, in fact, reached their highest levels in 15 years.
One key driver? A mismatch between circulating flu strains and this year’s vaccine. The dominant strains: H1N1 and H3N2, accounted for more than 99% of cases.
H3N2, in particular, is known for mutating quickly and evading immune responses, and only about half of circulating H3N2 samples matched well with vaccine antibodies, according to CDC surveillance data.
Another reason this year’s flu is hitting so hard: our immune systems are still catching up.
During the height of the COVID-19 pandemic, widespread masking, social distancing, and school closures suppressed not just SARS-CoV-2 but also seasonal flu and other common respiratory viruses. While that helped in the short term, it reduced community-level immunity over time, especially among children, who typically build natural resistance through repeated exposures.
“Young children who were toddlers or preschoolers during the pandemic missed early exposures to flu viruses,” explained experts at the nonprofit group Families Fighting Flu. “Now they’re in school, more socially active, and more vulnerable.”
The CDC reported a troubling spike in pediatric flu deaths this season with 216 fatalities, making it the deadliest flu season for children outside of a pandemic year. Neurological complications such as seizures and hallucinations also rose among young patients.
Another unusual trend: COVID-19 has taken a back seat this winter
Unlike previous years when COVID-19 variants dominated respiratory illness charts, flu has surged ahead as the top driver of doctor visits and hospitalizations. This could be due to a shift in viral dominance, changing weather patterns, or differences in immunity buildup. According to the CDC, nearly 8% of all outpatient visits are currently for flu-like symptoms, much higher than what’s typical for this time of year.
Vaccine Fatigue and Gaps in Coverage
Vaccination remains the strongest tool we have to fight influenza, but uptake has been stagnant, or worse, declining, in key groups.
As of April 2025:
Barriers like vaccine hesitancy, misinformation, racial and ethnic disparities in healthcare access, and fewer flu shot clinics in rural areas continue to widen the gap.
The 2025 flu has shown typical but often more intense symptoms than in previous years. Here’s what to look out for:
These symptoms may overlap with COVID-19 or RSV, but tend to come on faster and hit harder in flu cases this season.
For most healthy people, flu symptoms begin 1 to 4 days after exposure and typically last about 5 to 7 days. However, fatigue and cough may linger for up to two weeks.
You’re considered most contagious in the first 3 to 4 days after symptoms start but can continue to spread the virus up to a week later. The CDC recommends staying home until you’ve been fever-free for at least 24 hours without medication.
Vaccinated individuals may experience milder or shorter symptoms, but those with underlying conditions, young children, and older adults may have longer recoveries and higher risk of complications.
Yes, especially in people with weakened immune systems, chronic illnesses, or no prior flu immunity.
Possible complications include:
This is why experts stress that prevention remains the best medicine.
Here’s how to lower your risk during the remainder of the 2025 season:
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A lung cancer diagnosis shatters worlds. It thrusts patients into a whirlwind of fear, confusion, and uncertainty. What now? How will I cope? Is this the end? These questions are inevitable and urgent. Yet, amid the anxiety, many patients harbor a dangerous belief: that if cancer has taken hold, quitting smoking is pointless.
It's not just a myth, it’s dangerous. Newer studies show quitting smoking even after a cancer diagnosis can significantly improve survival, make treatments more effective, and alleviate symptoms. In other words: even after cancer strikes, letting go of cigarettes can save your life.
In many small towns across India, where awareness about cancer and tobacco risks is limited, patients often continue to smoke despite their diagnosis. “Some feel it is too late to stop. Others are too addicted or too hopeless to try,” says Dr. Ruchi Singh, HOD & Senior Consultant of Radiation Oncology at Asian Hospital. This is the kind of thinking that kills from the inside out.
The reality is the opposite. Dr. Singh emphasizes, “We try to explain … it is never too late. If they stop smoking, even after the cancer has started, the treatment becomes more effective. It is one of the most important things they can do for themselves.”
Every cigarette after diagnosis undermines treatment, weakens the body, and shortens survival. But should someone quit even late into their cancer journey their lungs begin to heal, treatments work better, and recurrence becomes less likely.
Global research aligns with Dr. Singh’s clinical advise, a study by IARC and Russian oncologists followed 517 lung cancer patients who smoked at diagnosis. Those who quit within three months lived 22 months longer on average and had 33% lower mortality risk and 30% lower disease progression, regardless of stage or smoking intensity.
The Prospective cohort of the Annals of Internal Medicine confirmed quitting after diagnosis yields meaningful survival benefits.
MUSC Hollings Cancer Center. A Harvard study of nearly 5,600 non-small cell lung cancer (NSCLC) patients found former smokers lived longer than current smokers, suggesting even pre-diagnosis quitting increases survival. Additional studies show quitting at or around diagnosis reduces mortality significantly and improves outcomes across all stages of NSCLC.
Smoking cessation isn’t just beneficial—it is one of the most powerful lifesaving interventions for lung cancer patients.
People tend to discount vaccines or preventive care because success makes the threat invisible. Lung cancer prevention has been a public health battle for decades. Policymakers and physicians worked to reduce smoking rates, and incidence fell. But once a cancer diagnosis arrives, all remission plans depend on a foundation of good habits—like quitting tobacco.
Tobacco smoke introduces toxins, weakens immune function, and diminishes treatment outcomes. Continuing to smoke after diagnosis:
Treatment regimens already overwhelm patients. Quitting smoking under stress and physical duress is tough—but not impossible. With the right support, patients dramatically increase their success odds. Here’s a compassionate roadmap:
Indeed, about 36% of tobacco-linked lung cancer patients manage to quit after diagnosis. Those are lives reclaimed.
Lung cancer still has a stigma. Many see it as self-inflicted. That stigma often delays help—including quitting support. But as Dr. Singh reminds us: “People think cancer means a death sentence. But many cases are treatable, especially if caught early. If someone quits smoking, we see real improvement such as better breathing, better recovery after surgery, and fewer chances of the cancer coming back.”
For patients, oncology teams, and families, smoking cessation after diagnosis isn’t optional—it’s urgent evidence-backed medicine.
A lung cancer diagnosis changes everything, but it does not define what comes next. Quitting smoking—even when the disease has already appeared—creates space for healing, response, and survival. It says, “I’m still here. I’m still fighting."
If you or someone you love is facing lung cancer- quit, today. It doesn’t erase the past—but it can extend the future. Numbers don’t lie: treatment plus quitting smoking can give us 22 more months, more energy, more peace, and a higher chance of beating this disease. Quitting is more than choice. It’s courage. And it is always worth it.
Credits: Health and me
When you stop for a moment, take a deep breath. How clean do you think is the air you just inhaled? If you live in a city or anywhere near heavy traffic, construction zones, or industrial areas chances are that breath carried more than just oxygen. Increasingly, health experts are raising a red flag: air pollution is becoming just as dangerous for your lungs as cigarette smoke. Unlike a lit cigarette, you can’t see it or smell it as easily, but the damage? It’s happening all the same—quietly, gradually, and across the globe.
On World Lung Cancer Day, it’s time to stop treating pollution as a background inconvenience and start seeing it for what it is: a major, modifiable threat to lung health.
We’ve long understood smoking as the leading cause of lung cancer, but the health narrative is shifting. Air pollution is catching up—and fast. According to the World Health Organization, over 7 million people die each year from air pollution, with billions more living with compromised respiratory health. Dr. Tedros Adhanom Ghebreyesus, Director-General of WHO, didn’t mince words back in 2018 when he said, “Air pollution is the new tobacco.”
The culprits are tiny, invisible particles like PM2.5 and PM10, nitrogen dioxide from vehicles, and toxins from burning biomass or fossil fuels. These pollutants don’t just irritate the lungs—they penetrate deep into lung tissue, triggering inflammation, reducing immune clearance, and increasing the risk of chronic respiratory illnesses and cancer. As the air gets dirtier, lung cancer in non-smokers—especially women and young adults—is on the rise.
We are already witnessing the consequences of environmental neglect warns Dr. Sachin Trivedi, Director of Medical Oncology at HCG ICS Khubchandani Cancer Centre, further adding, "Cigarette smoking has been known to be the major cause of lung cancer over the past decades. But there is a more recent and equally threatening danger that is on the rise: air pollution."
Dr. Trivedi highlights that a significant number of lung cancer diagnoses are now occurring in non-smokers, suggesting a stronger role for environmental pollutants. From vehicle emissions and industrial fumes to household fuel burning, the sources of this silent threat are everywhere. These pollutants infiltrate deep into the lungs, sparking chronic inflammation, oxidative stress, DNA damage, and even malignant mutations.
Even more concerning, these changes often don’t produce symptoms until the disease is advanced. Which is why early detection, lifestyle awareness, and pollution avoidance are critical. Dr. Trivedi urges individuals to recognize and act on subtle warning signs like chronic cough, shortness of breath, or unexplained weight loss, especially among non-smokers who may not suspect lung cancer.
Despite the scale of the problem, Dr. Trivedi emphasizes that it’s possible to shield your lungs through smart, consistent habits:
Especially in high-traffic or industrial areas, an N95 mask can block harmful particles like PM2.5. It’s a frontline defense your lungs will thank you for.
Ventilation is key. Use exhaust fans in kitchens, avoid indoor smoking, and install HEPA-filter air purifiers in high-pollution zones. Urban homes need this extra layer of air hygiene.
Track air quality through reliable apps. Skip rush hour outings and outdoor workouts when air quality is poor. Exposure control is protection.
What you eat matters. A diet high in vitamins A, C, and E from foods like berries, citrus fruits, broccoli, and nuts can counter oxidative lung damage. Turmeric and green tea also offer anti-inflammatory benefits.
A persistent cough or breathlessness isn’t always a passing cold. Get medical attention early—especially if you're a non-smoker experiencing unusual respiratory symptoms.
Dr. Devendra Parikh, Consultant in Surgical Oncology at HCG Aastha Cancer Centre, adds in a perspective, "Chronic polluted air harms our lungs just as smoking does: it silently, over time, injures delicate tissue and raises cancer risk. He stresses that fine particles from cooking smoke, traffic fumes, or even poorly ventilated homes carry microscopic toxins that inflame lung tissue and trigger genetic changes." He further shares more ways in which you can protect yourself
You can’t filter the world. But you can control your exposure, build resilient habits, and stay alert to what your lungs are telling you. The new reality is this: pollution is the new cigarette, and we can no longer afford to breathe without awareness. On World Lung Cancer Day and beyond—your breath is worth protecting. In cities where clean air isn’t guaranteed, your daily choices are your lungs’ best defense.
Pollution may feel as unavoidable as city noise, but it doesn’t have to be as destructive. By weaving these habits into your daily life, you give your lungs the best chance to clear toxins, reduce inflammation, and ward off the long-term threat of lung cancer.
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