Oncologists like Cathy Eng at Vanderbilt University Medical Center in Nashville, Tennessee and George Barreto, a surgeon from Mumbai, now at Flinders University in Adelaide, Australia are noticing a health trend, which has alarmed them and many other health experts. The trend in notice is about the types of cancers which are typically seen in older adults, but are now affecting younger people under 50.
Eng recalls a case of a 16-year-old with advanced gastrointestinal cancer, who flew from China to Texas but was too late by then. For Barreto, it was an observation among his friends and family that highlighted the rising incidence of early-onset cancers worldwide. He had promised that he would get to the bottom of this.
Statistics show that early-onset cancers, particularly colorectal, pancreatic, and stomach cancers, are increasing globally. The model suggests a 30% rise in cases by 2030.
While there is a decline in cancer deaths in some countries due to improved screening and treatment, early-onset cancers have now become more common. In the US, colorectal cancer is taking the lead as the cause of death in men under 50. There is a noticeable rise among women, Hispanic individuals, and American Indian and Alaska Native populations. Health disparities, including limited access to healthcare and systemic racism, also have become a catalyst in this trend, especially in marginalized communities.
There are multiple factors that could have led to the rise of early-onset cancer. Obesity, processed diets, alcohol consumption, and microbiome disruption are some of the most common ones.
For instance, changes in gut microbiota caused by diet or antibiotics can lead to inflammation and increase cancer risks. However, studies also show young, and seemingly healthy individuals developing aggressive cancers, this could indicate a more complex underlying reason.
Scientists are also looking at the genetic differences in early-onset tumors. Some aggressive tumors suppress immune responses more effectively than others. However, a conclusive finding is yet to come. Prenatal exposure to synthetic hormones is also being investigated. Some studies also aim to uncover environmental and lifestyle factors native to specific regions which could lead to such a condition. For instance, while Japan and South Korea are located near one another and have a similar topography and economy, the rate at which early-onset cancer is increasing in South Korea is must faster than in Japan.
The first and foremost thing to focus on would now be to increase screening and awareness. Advocacy campaigns are also targeting younger audiences and screening guidelines are being revised.
For instance, in the US, colorectal cancer screening now begins at 45, and Alaska Native health programmes have also recommended starting at 40.
The need for patient-centred care for young adults is of utmost importance. Cancer in younger patients can present unique challenges. Younger patients are also the ones who would want to prioritise fertility and long-term cognitive impact over treatment side effects. Many also face economic struggles, for instance, Candace Henley, who was diagnosed with colorectal cancer at 35 was raising five children. The aggressive surgery did not help her and left her unable to continue her job as a bus driver. She was left homeless. Physicians and the healthcare system must therefore understand the factors that can lead to more holistic care.
Researchers are suggesting a global biobank, where projects are kept to collect comprehensive data on prenatal and early-life exposures.
While unanswered questions still remain, progress is being made. Through advocacy and awareness campaigns, many patients are now seeking a second option, that is too, timely. Early detection and increased awareness are also helping many address the alarming rise in the rate of early-onset cancer, in the hope of solving the puzzle and getting more answers.
When strands clog your shower drain or your brush looks full every time you run it through your hair, panic is a natural response. Hair loss, or alopecia, is not just a cosmetic concern; it often hints at something deeper. And yet, thanks to internet half-truths and old wives’ tales, myths about alopecia spread faster than a viral meme. On World Alopecia Day, we turn to experts to separate fact from fiction while spotlighting the hidden medical conditions that might be behind the hair fall.
The good news? “Early diagnosis and treatment of these conditions can help restore hair growth and prevent permanent damage,” he assures.
Myth 1: “Only men experience alopecia.”
“While male pattern baldness is more commonly discussed, women are equally susceptible to alopecia due to hormonal imbalances, thyroid issues, and nutritional deficiencies,” says Dr Gangurde. Yes, ladies lose hair too and not just from brushing too hard.
Myth 2: “Stress alone is responsible for hair loss.”
While stress is definitely not good for your scalp’s health, it is not the lone cause. “Alopecia usually has multiple triggers, including genetics, autoimmune conditions, and underlying medical issues,” explains Dr Gangurde. Translation: stressing about stress causing hair fall might make things worse.
Myth 3: “Alopecia is always permanent and untreatable.”
This one might be the most damaging myth of all. “Many forms of hair loss, especially those caused by hormonal or nutritional factors, are reversible with timely medical intervention,” says Dr Gangurde. PRP therapy, medications, and lifestyle changes can all turn things around if you act early enough.
Do Not Just Shed Tears, Seek Help
If your hair has been thinning or falling out in clumps, resist the urge to DIY it with oils, serums, or social media hacks. “If you experience sudden or persistent hair loss, consult a dermatologist or trichologist promptly,” advises Dr Gangurde. “Early intervention can address the root cause, prevent progression, and in many cases, restore healthy hair growth.” Remember that alopecia is not just a surface-level issue. And with the right diagnosis, it is often more fixable than you think.
Credits: Canva
The 2025 flu season has turned out to be unlike any other in recent memory. This year, the flu season is marked by record-setting infections, multiple viral peaks, vaccine mismatches, and an overstretched healthcare system.
According to the Centers for Disease Control and Prevention (CDC), this year’s influenza activity is the most intense since the 2009 swine flu pandemic, with over 80 million estimated illnesses and rising. What’s driving this intensity, and what should the public know about prevention and symptom management?
Let’s break down what makes this flu season so severe and what it means for your health.
In most years, flu activity in the U.S. typically follows a predictable pattern, starting in October, peaking between December and February, and fading by April. But the 2024–2025 season has defied that rhythm.
Cases surged past the national baseline in December and then, unexpectedly, peaked again in February, a second wave that blindsided doctors and public health experts.
As of March 2025, the CDC had reported an estimated 37 million influenza infections, 480,000 hospitalizations, and 21,000 deaths. Hospitalizations, in fact, reached their highest levels in 15 years.
One key driver? A mismatch between circulating flu strains and this year’s vaccine. The dominant strains: H1N1 and H3N2, accounted for more than 99% of cases.
H3N2, in particular, is known for mutating quickly and evading immune responses, and only about half of circulating H3N2 samples matched well with vaccine antibodies, according to CDC surveillance data.
Another reason this year’s flu is hitting so hard: our immune systems are still catching up.
During the height of the COVID-19 pandemic, widespread masking, social distancing, and school closures suppressed not just SARS-CoV-2 but also seasonal flu and other common respiratory viruses. While that helped in the short term, it reduced community-level immunity over time, especially among children, who typically build natural resistance through repeated exposures.
“Young children who were toddlers or preschoolers during the pandemic missed early exposures to flu viruses,” explained experts at the nonprofit group Families Fighting Flu. “Now they’re in school, more socially active, and more vulnerable.”
The CDC reported a troubling spike in pediatric flu deaths this season with 216 fatalities, making it the deadliest flu season for children outside of a pandemic year. Neurological complications such as seizures and hallucinations also rose among young patients.
Another unusual trend: COVID-19 has taken a back seat this winter
Unlike previous years when COVID-19 variants dominated respiratory illness charts, flu has surged ahead as the top driver of doctor visits and hospitalizations. This could be due to a shift in viral dominance, changing weather patterns, or differences in immunity buildup. According to the CDC, nearly 8% of all outpatient visits are currently for flu-like symptoms, much higher than what’s typical for this time of year.
Vaccine Fatigue and Gaps in Coverage
Vaccination remains the strongest tool we have to fight influenza, but uptake has been stagnant, or worse, declining, in key groups.
As of April 2025:
Barriers like vaccine hesitancy, misinformation, racial and ethnic disparities in healthcare access, and fewer flu shot clinics in rural areas continue to widen the gap.
The 2025 flu has shown typical but often more intense symptoms than in previous years. Here’s what to look out for:
These symptoms may overlap with COVID-19 or RSV, but tend to come on faster and hit harder in flu cases this season.
For most healthy people, flu symptoms begin 1 to 4 days after exposure and typically last about 5 to 7 days. However, fatigue and cough may linger for up to two weeks.
You’re considered most contagious in the first 3 to 4 days after symptoms start but can continue to spread the virus up to a week later. The CDC recommends staying home until you’ve been fever-free for at least 24 hours without medication.
Vaccinated individuals may experience milder or shorter symptoms, but those with underlying conditions, young children, and older adults may have longer recoveries and higher risk of complications.
Yes, especially in people with weakened immune systems, chronic illnesses, or no prior flu immunity.
Possible complications include:
This is why experts stress that prevention remains the best medicine.
Here’s how to lower your risk during the remainder of the 2025 season:
Credits: Canva
A lung cancer diagnosis shatters worlds. It thrusts patients into a whirlwind of fear, confusion, and uncertainty. What now? How will I cope? Is this the end? These questions are inevitable and urgent. Yet, amid the anxiety, many patients harbor a dangerous belief: that if cancer has taken hold, quitting smoking is pointless.
It's not just a myth, it’s dangerous. Newer studies show quitting smoking even after a cancer diagnosis can significantly improve survival, make treatments more effective, and alleviate symptoms. In other words: even after cancer strikes, letting go of cigarettes can save your life.
In many small towns across India, where awareness about cancer and tobacco risks is limited, patients often continue to smoke despite their diagnosis. “Some feel it is too late to stop. Others are too addicted or too hopeless to try,” says Dr. Ruchi Singh, HOD & Senior Consultant of Radiation Oncology at Asian Hospital. This is the kind of thinking that kills from the inside out.
The reality is the opposite. Dr. Singh emphasizes, “We try to explain … it is never too late. If they stop smoking, even after the cancer has started, the treatment becomes more effective. It is one of the most important things they can do for themselves.”
Every cigarette after diagnosis undermines treatment, weakens the body, and shortens survival. But should someone quit even late into their cancer journey their lungs begin to heal, treatments work better, and recurrence becomes less likely.
Global research aligns with Dr. Singh’s clinical advise, a study by IARC and Russian oncologists followed 517 lung cancer patients who smoked at diagnosis. Those who quit within three months lived 22 months longer on average and had 33% lower mortality risk and 30% lower disease progression, regardless of stage or smoking intensity.
The Prospective cohort of the Annals of Internal Medicine confirmed quitting after diagnosis yields meaningful survival benefits.
MUSC Hollings Cancer Center. A Harvard study of nearly 5,600 non-small cell lung cancer (NSCLC) patients found former smokers lived longer than current smokers, suggesting even pre-diagnosis quitting increases survival. Additional studies show quitting at or around diagnosis reduces mortality significantly and improves outcomes across all stages of NSCLC.
Smoking cessation isn’t just beneficial—it is one of the most powerful lifesaving interventions for lung cancer patients.
People tend to discount vaccines or preventive care because success makes the threat invisible. Lung cancer prevention has been a public health battle for decades. Policymakers and physicians worked to reduce smoking rates, and incidence fell. But once a cancer diagnosis arrives, all remission plans depend on a foundation of good habits—like quitting tobacco.
Tobacco smoke introduces toxins, weakens immune function, and diminishes treatment outcomes. Continuing to smoke after diagnosis:
Treatment regimens already overwhelm patients. Quitting smoking under stress and physical duress is tough—but not impossible. With the right support, patients dramatically increase their success odds. Here’s a compassionate roadmap:
Indeed, about 36% of tobacco-linked lung cancer patients manage to quit after diagnosis. Those are lives reclaimed.
Lung cancer still has a stigma. Many see it as self-inflicted. That stigma often delays help—including quitting support. But as Dr. Singh reminds us: “People think cancer means a death sentence. But many cases are treatable, especially if caught early. If someone quits smoking, we see real improvement such as better breathing, better recovery after surgery, and fewer chances of the cancer coming back.”
For patients, oncology teams, and families, smoking cessation after diagnosis isn’t optional—it’s urgent evidence-backed medicine.
A lung cancer diagnosis changes everything, but it does not define what comes next. Quitting smoking—even when the disease has already appeared—creates space for healing, response, and survival. It says, “I’m still here. I’m still fighting."
If you or someone you love is facing lung cancer- quit, today. It doesn’t erase the past—but it can extend the future. Numbers don’t lie: treatment plus quitting smoking can give us 22 more months, more energy, more peace, and a higher chance of beating this disease. Quitting is more than choice. It’s courage. And it is always worth it.
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