Telling people to “eat less and move more” isn’t working. If it were, we wouldn’t be facing a growing obesity crisis, staggering healthcare costs, and lives eroded by stigma and chronic illness. The problem isn’t individual failure it’s systemic.

Obesity is real, chronic, and deeply rooted in our biology, environment, and society. Yet for years, policy responses have focused on personal responsibility, leaving millions to fend for a solution that only lasts till the next trip to the grocery store. To truly address this crisis, everything from individual care to global food systems must changes.

We’ve been sold the myth that weight gain is simply a matter of willpower and to eat less, move more. But emerging science tells a different story- obesity is a chronic relapsing condition, driven by a tangle of genetic, psychological, socioeconomic, and environmental forces.

In England, 26.5% of adults live with obesity. Among children aged 10–11, that number is 22.1%. Beyond fat and numbers, obesity carries heavy consequences: heart disease, type 2 diabetes, poor mental health, and more. It is estimated to cost the UK £126 billion annually—covering NHS services, unemployment, lost quality of life, and informal care support. And it’s only getting worse; by 2035, this could rise to £150 billion a year.

It’s time to recognize obesity as more than a lifestyle issue. It’s a disease born in context and reinforced by neglect.

Is The “Obesogenic” Environment Built Just for Weight Gain?

In 2007, the UK’s Foresight report introduced “obesogenic environment”—a world where high-calorie ultra-processed foods are cheap and ubiquitous, and physical activity is less necessary than ever. This isn’t accidental—it’s engineered.

Our cities encourage driving over walking. Junk food dominates retail shelf space. Work and leisure revolve around screens. In many neighborhoods, healthy options are scarce, thanks to food deserts, poor public transit, and lack of green space.

These aren’t universal conditions. Communities with lower income face disproportionately higher exposure to these obesogenic environments. That biological response to unhealthy surroundings is not a failing—it’s expected.

It’s easy to shift blame onto individuals. But telling someone to lose weight without addressing the systemic obstacles is like asking someone to swim upstream while you drain the river.

Weight stigma thrives in this climate. Without understanding obesity's roots, people struggling with weight are often labeled as lazy or undisciplined. That stigma breeds shame and discourages seeking care—especially childhood, when a lifetime of self-consciousness can begin but data shows the real drivers: deprivation correlates with rising obesity rates, especially among kids. Our approach must evolve from blame to empathy and evidence-based support.

Why it Is Important to Reframe Obesity?

To create meaningful change, we must dismantle three harmful myths:

• Obesity is a personal failure.
• Weight loss is about willpower alone.
• Short-term diets and exercise plans are solutions.

Instead, care must be rooted in medical science and social context. Here’s how:

1. Recognize Obesity as a Disease

Health systems need to treat obesity like chronic conditions—diabetes, asthma, depression—not as an afterthought. This means consistent assessment, structured interventions, and multi-year follow-ups.

2. Confront Weight Stigma

Weight-based bias is tolerated in schools, workplaces, and clinics. This must stop. Clinicians and educators require training to adopt respectful, person-centered language and practices. Public health campaigns should back this shift away from blame.

3. Deliver Tailored, Holistic Care

No one-size-fits-all. Treatment plans must consider genetics, culture, environment, mental health, and lifestyle. Follow-up must be more than numbers on a scale. Shared decision-making, psychological support, and mindful goal-setting are critical.

4. Change Surrounding Environments

To make healthy living feasible, the environment must support it. That means:

• Expanding the sugar tax to all sugary beverages and high-sugar foods.
• Reformulating ultra-processed foods to reduce calories, salt, fat, and sugar.
• Mandating clear labelling and restricting advertising of unhealthy foods, especially to children.
• Investing in public transit, affordable fresh food, and accessible parks, especially in underserved areas.
• Implementing workplace health-friendly policies—standing desks, active transport rewards, fresh food in cafeterias.

This isn’t about personal choice—it’s about choice architecture.

What Are The Economic, Social, and Ethical Stakes Of obesity Crisis?

The cost of obesity isn’t only financial. There’s a social toll: family stress, workplace discrimination, mental health crises. Governments are waking up to the reality: ignoring systemic factors isn’t just irresponsible—it’s reckless.

Expanding sugar taxes alone could raise billions annually, reduce consumption, and fund essential public services. When coupled with broader reforms, these policies can shift cultural norms and industry incentives.

Moreover, recognizing socioeconomic factors—housing, education, income inequality—changes the narrative. It places the problem in context, not on individuals already struggling to stay afloat.

Everyday Steps That Need Redefining

A systems approach doesn’t erase personal responsibility—it reframes it. People matter in the solution, but environments and systems matter more. Imagine a world where:

• Free nutrition counseling is available during pediatric checkups.
• Primary care physicians routinely screen for weight stigma and mental health.
• Urban planners make biking as safe and common as driving.
• Grocery stores compete based on healthy food affordability.
• Schools offer fresh meals, cooking classes, and daily movement.
• Employers design breaks and infrastructure to support employee wellness.

That’s not future talk—it’s policy within reach. These aren’t random ideas—they’re pieces of an integrated solution.

Why We Need the Shift Now?

We face a choice: continue business-as-usual and watch costs and suffering escalate, or embrace a comprehensive public health approach. The evidence is clear. Individual change without systemic transformation is a drop in the bucket.

Treating obesity as a chronic disease, ending stigma, delivering personalized support, and redesigning our environments represent a paradigm shift. And yes—it will require political will, public will, and significant change in how we talk about weight but the payoff is enormous with lives spared, healthcare systems stabilized, and children unburdened by preventable disease and shame. It’s not just about waistlines—it’s about equity and human dignity.

If the goal is to support healthier populations—and healthier systems—then blaming individuals for obesity is both harmful and counterproductive. The “eat less, move more” era is over.

To tackle obesity, we must reframe it from a personal failing to a collective challenge. That means shifting from judgment to understanding, from short-term fixes to long-term systems. From individual blame to societal accountability because real health isn’t about individual transformation alone—it’s about transforming the world we live in.

Blackmores Vitamin B6 Case: "I first reported numb hands and feet to doctors in 2012, when I was taking vitamin B6 in various supplements. In 2019, I suffered a paralysed vocal cord, which affects my ability to speak and swallow liquid. I was teaching English at TAFE and I basically had to give up my job because I could not speak. I had other symptoms, like stabbing in my feet, numbness, headaches, and gut pain. I have seen dozens of specialists over he past 10 years, trying to get a diagnosis on my "mystery illness"," shares 61-year-old, Penny Thompson from Wollongong to The Sydney Morning Herald.

There are many others like her. The reason behind all this is the over-the-counter vitamins. The Sydney Morning Herald reports that many patients developed twitches, migraines, nerve damages, and other neurological symptom after they took over-the-counter supplements. These are the same patients who have now "joined a class action investigation against wellness giant Blackmores", for selling vitamin "linked to a growing number of toxicity cases".

What Is Happening With OTC Vitamins?

In May, Melbourne injury law firm Polaris announced that it was pursuing a class action investigation against Blackmores, an Australian health supplement company, on behalf of anyone who has suffered injuries after taking its supplements. The reason being, it contains "higher than recommended levels of vitamin B6".

The law firm's principal, Nick Mann, said that they have more than 300 inquiries joining the potential suit. Majority of these respondents have suffered injuries after consuming Blackmore's supplements that contain vitamin B6.

“One of the things that surprised me has been the number of people who’ve told us about really significant and ongoing impacts of B6 toxicity long after they’ve ceased taking the supplement,” Mann told The Sydney Morning Herald. “The impacts on them have been permanent and ongoing.”

However, Blackmores spokesperson told the media outlet that all its products including those that contain vitamin B6 are "in strict accordance" with Therapeutic Goods Administration's (TGA), a regulatory agency of the Australian Government as part of its Department of Health, Disability and Ageing, regulatory requirement.

Is Vitamin B6 Toxic?

Vitamin B6 is considered toxic when consumed in high doses over a long period. However, the TGA notes that there was no scientific consensus on a 'safe' threshold.

The TGA notes: "Taking vitamin B6 even at low doses can cause peripheral neuropathy but people are more likely to get it if they are taking more than one supplement."

A 2022 report by the TGA notes that it has receieved over 30 reports of peripheral neuropathy, which are related to products containing vitamin B6.

The Netherlands Pharmacovigilance Centre Lareb has also previously reported on nerve damage caused by the use of nutritional supplements that contain vitamin B6.

As per Lareb's 2024 report, "A healthy and varied diet will provide most people with enough vitamin B6. Yet many people take nutritional supplements with vitamin B6 in addition to their regular diet. Excessive intake of vitamin B6 over a long period of time can sometimes lead to damage to nerves in arms and/or legs. Symptoms are tingling, numbness or pain in hands and/or feet."

What Is The Recommended Levels of Vitamin B6?

Lareb notes that the recommended dietary allowance for adults is 1.5 milligram of vitamin B6 per day. Whereas, the TGA notes that maximum permitted daily dose in individual products has been reduced to 100mg for adults and even less for children. However, peripheral neuropathy can occur at very low doses, which could be less than 50mg.

This is not just the case with vitamin B6, but consuming any vitamin without being prescribed could lead to nutrient overdose and cause blood clots, and/or infections. Dr Shrey Kumar Srivastav, Senior Consultant, Internal Medicine at Sharda Hospital says, "frequent or routine use can cause vitamin toxicity or electrolyte imbalances". While some people opt for oral supplements, others opt for IV supplements. However, Dr Prashant Sinha, Head of Emergency and Internal Medicine at PSRI Hospital, Delhi, says that for a healthy person with no diagnosed deficiency, there is generally no medical reason to receive IV supplements regularly. "Getting them too frequently can lead to nutrient overload or dependency, where the body becomes less efficient at natural nutrient absorption," he points out.

ALSO READ: The Truth About IV Drips: Are They Better Than Food Or Supplements?

Symptoms To Look Out For:

• Nausea
• Hypersensitivity
• Nerve damage could lead to tingling, burning, and numbness throughout the body

A recent study suggests that eating eggs may be linked to a lower risk of developing Alzheimer’s disease. The study, published in The Journal of Nutrition, found that individuals who consumed eggs regularly had significantly reduced levels of the brain protein buildup typically associated with the condition.

The researchers went beyond just surveys and dietary recall; they examined brain tissue from autopsies to get a clearer picture of what was going on inside. And what they found was surprising. Individuals who ate eggs more frequently had less of the sticky, toxic protein buildup in the brain, the kind typically associated with Alzheimer’s. In fact, those who ate just one egg a week had a 47 per cent lower risk of developing the disease compared to people who rarely touched the yolk.

Nutrient Rich

This might come as a shock to those who still see eggs as little cholesterol bombs. But scientists are now pointing fingers at something far more benevolent than blameworthy: eggs’ high choline and omega-3 content.

Choline is an essential nutrient that plays a crucial role in brain development and memory function. It also helps form acetylcholine, a neurotransmitter involved in mood, muscle control, and cognitive performance. Omega-3 fatty acids, meanwhile, have long been praised for their brain-protective properties, including reducing inflammation and supporting cell membrane health.

Together, these nutrients could be helping to keep those harmful proteins from building up in the brain, the very proteins that leave a damaging mark in Alzheimer’s patients.

From Kitchen Staple to Cognitive Support

Eggs have been a part of the human diet for millennia. They are easy to prepare, affordable, and versatile enough to land on breakfast, lunch, or dinner plates. This latest research invites us to reconsider eggs not just as a protein-packed breakfast option but as a strategic ally in long-term brain health.

A Word of Caution

It is important to note that this was an observational study. It shows an association, not cause and effect. But it does add weight to the growing body of research that supports the role of specific nutrients in cognitive protection.

The study’s authors emphasise that more work needs to be done to fully understand the mechanisms at play. Still, they suggest that incorporating eggs into a balanced diet could be a simple and accessible way to help support brain function as we age.

You have had a long day, maybe your inbox is overflowing, you argued with your partner, or you are actually feeling unwell. Before you know it, you’re elbow-deep in a bag of chips or reaching for that extra scoop of ice cream. Sound familiar?

You are not the only one and it’s not just about willpower. Emotional eating, especially the urge to reach for comfort food during stress, is a real, biological phenomenon. And science is now helping us understand exactly how stress rewires the brain to seek food—not for nourishment, but for relief.

How Stress Hijacks Your Brain and Appetite?

Stress affects nearly every system in your body, but it’s your brain that calls the shots when it comes to hunger. A new study published in the journal Neuron offers powerful insight into how chronic stress interferes with the brain’s natural appetite regulation system.

At the center of it is the lateral habenula, a region of the brain responsible for registering when you’ve had enough to eat. Under normal circumstances, this part of the brain sends “stop” signals—telling you that you're full, satisfied, and can put the fork down but under chronic stress? Those signals get muffled.

According to senior author Dr. Herbert Herzog, stress can “override a natural brain response that diminishes the pleasure gained from eating,” which essentially means the brain keeps rewarding you for eating—long past the point of physical hunger.

In lab experiments, stressed mice were observed to keep eating high-fat foods without reaching satiety, and they even consumed up to three times more sweeteners like sucralose. Their brains released NPY (neuropeptide Y), a molecule that fuels cravings and weight gain. When researchers blocked NPY’s action, the mice consumed less—and gained less weight.

Why Do We Crave Comfort Food When Stressed?

Comfort food is rarely about comfort. It’s about dopamine. When you're stressed, your body releases cortisol and adrenaline, preparing for a fight-or-flight response. In the short term, this can suppress appetite but when stress becomes chronic—weeks, months, years—it starts having the opposite effect. Cortisol ramps up your desire for high-fat, high-sugar foods that light up the brain’s reward center.

This explains why you're not stress-craving a salad. You want cookies, pasta, chocolate. These foods activate dopamine—the brain's feel-good chemical—providing temporary emotional relief. But the cycle is damaging. The more you indulge to cope, the harder it becomes to stop, and the more the brain links eating with emotional regulation.

Long-Term Effect of Chronic Stress and Emotional Eating

It’s not just about a few extra pounds. Prolonged emotional eating can lead to serious health consequences:

• Obesity and metabolic syndrome
• Type 2 diabetes
• High blood pressure and heart disease
• Hormonal imbalances
• Chronic inflammation and oxidative stress

Cortisol doesn’t just impact appetite. Over time, it can damage organs and impair your body’s ability to regulate inflammation and oxidative stress—both key factors in chronic disease.

Emotional Eating vs. Real Hunger: How To Tell The Difference?

One of the simplest tools to fight stress eating is to pause and ask: Am I actually hungry?

Physical hunger builds gradually and is felt in the body—your stomach growls, you feel a bit low on energy. Emotional hunger, by contrast, strikes suddenly and is usually hyper-specific (think: “I need fries right now”). It’s often driven by boredom, sadness, anger, or anxiety, not a true need for fuel.

Simple Tips to Break the Stress-Eating Cycle

Overcoming emotional eating isn’t about shame—it’s about strategy. Here are practical, science-backed ways to regain control:

1. Manage Stress at Its Root

Meditation, yoga, deep breathing, or even a quick walk outside can lower cortisol and help you regulate mood before turning to food.

2. Start a Food and Mood Journal

Writing down what you eat and how you feel before and after helps identify patterns. Are you really hungry—or just stressed, sad, or bored?

3. Restructure Your Environment

Keep comfort foods out of immediate reach. Stock your fridge and pantry with healthy options—fresh fruits, vegetables, whole grains, nuts—so if a craving hits, you have better options.

4. Pause Before You Snack

Next time you feel the urge to snack outside of mealtime, take a breath. Set a 10-minute timer. Often, the craving fades or changes.

5. Choose Nourishment, Not Numbing

If you need to eat, fine—but choose something that sustains you: a banana with nut butter, a bowl of oats, a handful of almonds. These foods keep your blood sugar steady and mood stable.

6. Build an Emotional Toolkit

Food can’t be your only coping strategy. Call a friend, do a puzzle, take a bath, journal, or listen to music. Emotional regulation isn’t about avoidance—it’s about redirection.

Here’s what no one tells you- stress eating is deeply human. It’s a biological reflex tied to emotional needs. But once you understand what’s happening in your brain and body, you can start to break the loop.

Awareness is the first step. From there, it’s about replacing automatic reactions with intentional responses—choosing to nourish your body instead of numbing your emotions.

Cravings may still come and go but now, you’ll know where they’re coming from—and how to rise above them.

Your brain isn’t broken, it’s just responding to stress the way it’s wired to. You have more power than you think—to eat with intention, manage stress better, and reclaim your health one mindful choice at a time.