Credits: Canva
A newly emerging “untreatable” mystery virus that is being described as stronger than Covid is now spreading across several parts of the world, with experts warning that even common disinfectants may not be effective against it. Known as adenovirus, the infection causes symptoms similar to a severe bout of flu, such as shortness of breath, a runny nose, and a sore throat. What sets it apart, however, is the limited treatment available.
In most cases, people who contract the virus have no option but to manage symptoms and allow the illness to pass on its own. The good news is that adenovirus infections are usually mild. That said, much like Covid or seasonal flu, the risk rises for people with weakened immune systems, who may experience more serious complications, according to a report by The Mirror.
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Jefferson Health’s medical director of infection prevention and control, Eric Sachinwalla, has cautioned that unlike more familiar viral infections, there is very little doctors can do to actively treat adenovirus.
Adenoviruses belong to a broad family of common viruses that can affect multiple parts of the body, including the airways and lungs, eyes, digestive system, urinary tract, and even the nervous system. They are a frequent cause of fever, cough, sore throat, diarrhoea, and conjunctivitis. Most infections tend to be mild and clear up on their own within a few days. However, health experts are now noting a rapid rise in cases, with the virus spreading quickly and leaving large numbers of people unwell.
The virus is particularly contagious because it is tougher than many others. Routine cleaning with soap and water or standard disinfectants may not be enough to eliminate it, allowing it to survive longer in the environment. This is why outbreaks are often seen in places such as day-care centres and military barracks, where close contact is common. Adenovirus spreads through respiratory droplets, can be passed through stool, and can linger on contaminated surfaces for extended periods, as per Mirror.
Symptoms of adenovirus infection can vary widely. Common signs include shortness of breath, a runny nose, and a sore throat. Some people may also develop diarrhoea or pink eye. The wide range of symptoms is partly due to the fact that there are more than 60 known strains of the virus.
Adenoviruses, like coronaviruses, spread from person to person and can trigger similar respiratory symptoms. However, they belong to entirely different virus families and behave differently. One key difference is resistance. Coronaviruses are more easily destroyed by disinfectants, while adenoviruses are harder to kill, which allows them to spread more easily than Covid or flu.
For otherwise healthy individuals who feel unwell but do not have severe symptoms such as high fever or breathing difficulty, recovery usually happens at home with basic supportive care. Medical attention is more important for people with weakened immunity, parents of very young infants, or those with existing conditions like heart or lung disease. If symptoms appear, experts advise against walking straight into a clinic. Calling ahead is safer, as doctors may recommend a telehealth consultation if the illness seems highly contagious.
Most adenovirus infections are mild and resolve without medical treatment. However, if symptoms linger or worsen, there is often little doctors can do beyond monitoring and symptom relief, as the virus largely needs to run its course. Following basic hygiene measures, such as washing hands regularly and cleaning frequently touched surfaces, remains one of the most effective ways to reduce the risk of infection.
Credits: AP
Though Charlie Sheen’s HIV has been described as “completely manageable,” the actor recently shared that he once came across a treatment he believed worked far better than existing options, but it never reached the public. Speaking on the *Howie Mandel Does Stuff* podcast, the 60-year-old actor reflected on an experimental drug he used years ago and explained why it ultimately disappeared from view. “There was one that was really good that I was hoping would come to market one day, and it never did,” said Sheen, who publicly disclosed his HIV diagnosis in 2015. This has raised a key question: which experimental drug is Charlie Sheen referring to?
Human immunodeficiency virus, or HIV, is a virus that attacks the immune system. If left untreated, it can progress to acquired immunodeficiency syndrome, or AIDS, which represents the most advanced stage of infection. HIV primarily targets white blood cells, weakening the body’s natural defences. The virus spreads through unprotected sexual contact, sharing needles for drug use, exposure to infected blood, and from parent to child during pregnancy, delivery, or breastfeeding.
HIV infects CD4 cells, also known as helper T cells, which play a central role in immune response. As the virus destroys these cells, the white blood cell count drops, leaving the body vulnerable to infections it would normally fight off with ease.
Early on, HIV can cause flu-like symptoms. After that, it may remain hidden in the body for years without obvious signs, while continuing to damage the immune system. When CD4 levels fall very low, or when certain serious infections develop, HIV is considered to have progressed to AIDS.
At this stage, symptoms may include rapid weight loss, severe fatigue, sores in the mouth or genitals, recurring fevers, night sweats, and changes in skin colour.
During the podcast conversation, Sheen named the drug he believes made a major difference. “That was a thing called PRO 140,” he said. He described it as a monoclonal antibody that produced faster and more consistent results, with fewer side effects than standard treatments. When asked why it never became widely available, Sheen suggested it may have posed a threat to existing therapies. “It works, better than what they have,” he said, adding that the company behind it ran into serious trouble. Mandel responded that someone should investigate further, a point Sheen agreed with.
PRO 140 is a humanised monoclonal antibody designed to block CCR5, a receptor HIV commonly uses to enter human cells and replicate. Around 70 percent of people living with HIV in the United States, and as many as 90 percent of newly diagnosed patients, carry CCR5-tropic strains of the virus.
Earlier studies found that a single intravenous dose of PRO 140 sharply reduced HIV levels, while weekly injections under the skin lowered viral load much more effectively than a placebo. Research also suggested that the drug did not interfere with normal immune functions linked to CCR5.
According to Aidsmap, PRO 140 was generally considered safe and well tolerated. Although more than 90 percent of participants in extended studies reported side effects, there were no serious adverse reactions linked directly to the drug, and no one had to stop treatment because of it. The reported side effects were limited to injection-site reactions, which were usually mild to moderate.
Sheen has repeatedly spoken about his experience with the drug, maintaining that it delivered steadier results and caused fewer side effects than conventional HIV treatments.
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Weight loss drugs are now common, but so are its side effects, especially when you stop taking it. There are numerous reports of how these popular weight loss jabs like Ozempic, Mounjaro, and Wegovy, once stopped, had changed people's cravings, food choices, and some have reported that weight too have come back.
However, Dr Donald Grant, GP and senior clinical advisor at The Independent Pharmacy tells Metro that it is quite common for people to regain some of the weight once they stop the medication. As of now, 1.5 million people in the UK are using these jabs, and reports say that some may end up heavier than when they started. Why does this happen?
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Dr Grant says that there are various factors including metabolism to appetite, however, there are also 7 common mistakes that people do once they stop taking their weight loss jabs, which must be avoided.
Going cold turkey could increase the weight, warns Dr Grant. Since these medicines work by suppressing the appetite, stopping the medications suddenly could lead to poor eating habits and potential weight gains. The best way? Talk to your GP before making a decision to come off the weight loss drugs. A medical professional could guide you on how to leave the medication, while making amends to your lifestyle habits.
A recent study by scientists at the Oxford University, of the 6,000 people who use GLP-1 drugs, most will gain back their weight within the 10 months of stopping the medicine. This happens because once the medicines are stop, people do not replace the treatment with healthy habits. this could lead to weight gain.
Doctors suggest that even 30 minute of daily exercise could make the difference and burn the extra calories.
If you stop taking your GLP-1 medications early, it won't work. Dr Grant points out that this is one of the most common mistakes people make. "These drugs are designed to be used long-term, but not doing so gives the body little time to adapt to the treatment."
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If you continue eating unhealthy while you are on Ozempic, Mounjaro or Wegovy, then you will regain the weight once you stop. Dr Grant says, "It is important for people to draw up a dietary plan, including structured meal times to avoid overeating. I also recommend a well-balanced diet including a variety of high-protein, fibre-rich and healthy fat food."
"While it’s important not to create an unhealthy relationship with weight management, tracking weight closely once ending any treatments can help people adjust their routine to avoid losing progress," says Dr Grant.
Regular monitoring weight can ensure that you can get back on track before you lose too much progress after you stop taking your weight loss drugs.
If you have an emotional relationship with your food, then weight gain is possible. This is because emotional eating could lead to hormonal imbalance, stress, poor sleep, and unresolved trauma. While the jab can reduce weight, once stopped, if emotional eating is continued, you can easily gain it back. The best way to deal with this is by addressing the issue, speaking to an expert or a mental health specialist.
Even after you stop your weight loss drugs, it is important that you continue eating enough protein. Protein helps in muscle building. Not eating enough protein, could lead to weight gain, by muscle loss and lower metabolism.
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"You are 35; you are healthy. It is probably a panic attack," said Kristina Auwarter, as reports SELF Magazine, when she first felt a throbbing pain in her chest when she was picking her son up from his crib. There was nothing to be worried about she thought because she had been working out, her blood work was fine. She thought it was just a heartburn and popped Tums. Had it not been her sister who was home that day, she wouldn't have called 9-1-1. When she reached at the hospital, she learned what she had was a heart attack. She learned that she had a total blockage in the largest of the three arteries that feed the heart.
She is far from someone who would get a heart attack. However, a 2018 study published in AHA|ASA Journals show that there had been a surge in younger patients, under 54 for heart attack between 1995 and 2004, and it has led to a 10% jump among women being hospitalized for the same, while the proportion of men remained the same. Another 2019 research by the American College of Cardiology reported that people under age of 50 were reporting more heart attacks, and the number had jumped to 2% each year from 2000 to 2016.
For women, the long-term impact of a heart attack at a young age is often more severe. Multiple studies show that younger women are less likely than men to receive timely tests and appropriate treatments, and are more likely to be re-hospitalized or die later from heart disease.
Hormonal conditions like polycystic ovary syndrome (PCOS) can accelerate risk factors such as high blood pressure, high cholesterol, and elevated blood sugar. All of them could fuel plaque buildup in the arteries. This has become even more common due to less nutritious diets and increasingly sedentary lifestyle.
While these are some of the traditional risk factors, non-traditional risk factors often disproportionately affect young women. These include adverse pregnancy outcomes such as hypertensive disorders of pregnancy, including preeclampsia, and gestational diabetes. Rates of pregnancy-related high blood pressure have doubled over the past two decades, while gestational diabetes has climbed by roughly 30%, trends likely tied to worsening pre-pregnancy health and lifestyle shifts that are not particularly heart-friendly.
The matter of the fact is that for the longest these conditions were viewed as temporary problems, confined to pregnancy. The assumption was, one a person delivered, the dangers passed, however, it is not the case. There are numerous research that suggest that these complications can double and even more than double the risk of future cardiovascular diseases, including heart attack. Scientists have suspected that they may either reveal an underlying vulnerability to heart disease or trigger lasting inflammation or damage to blood vessels.
Mental health is another underappreciated piece of the puzzle. Women are about twice as likely as men to experience mental health conditions, and that disparity carries heart-related consequences. SELF reports that women with depression face a higher risk of developing cardiovascular disease than men with the same diagnosis, and psychological distress appears to raise future heart risk in women but not men. Researchers believe women may experience more intense mental health symptoms or a stronger biological stress response, which could translate into greater strain on the heart over time.
Autoimmune diseases add yet another layer of risk. These conditions, which are roughly twice as common in women, are characterized by chronic inflammation. Over years, that inflammation can damage the lining of blood vessels, quietly increasing the likelihood of a heart attack.
The biggest issue is that many of these atypical risk factors are not included in the standard tools doctors use to estimate heart attack risk. As a result, opportunities to intervene early are often missed, particularly in younger women. In one study of 3,500 young people who experienced a heart attack, women were significantly less likely than men to recall a doctor ever discussing their heart disease risk.
This gap in awareness carries over to diagnosis and treatment. Because heart attacks are still widely stereotyped as an older man’s problem, young women may not recognize what is happening when symptoms appear, even when those symptoms include classic chest pain. At the same time, the message that women often have “different” heart attack symptoms can backfire, leading some to dismiss chest discomfort altogether. Both things can be true: chest pain or pressure remains the most common symptom across sexes, but women are also more likely to experience additional, less typical signs.
These can include pain or tightness anywhere from the jaw to the abdomen, shortness of breath, sweating, nausea, unusual fatigue, or a vague sense that something just isn’t right. For many women, the sensation is not dramatic or crushing, just unfamiliar, which makes it easier to ignore. Combine that with the reality that women’s symptoms are more likely to be downplayed or attributed to anxiety or stress, and delays in seeking care become almost inevitable. Research cited by SELF even shows women are more likely to call an ambulance for a male partner than for themselves.
Diagnosis becomes even trickier when a heart attack is triggered by something other than the classic plaque buildup in the arteries—a scenario that appears to be far more common in younger women. A 2025 Mayo Clinic study found that more than half of heart attacks in women under 65 were caused by nontraditional mechanisms such as blood clots traveling to the heart, artery spasms, or spontaneous coronary artery dissection (SCAD), compared with about a quarter of cases in men. SCAD, in particular, overwhelmingly affects women.
Doctors are still unraveling why these atypical heart attacks skew female, but theories point to a mix of genetics, differences in blood vessel structure, hormonal fluctuations, and the effects of intense physical or emotional stress. Because these events are not driven by plaque, they can strike women who have none of the classic risk factors, making them easier to miss and harder to diagnose in time.
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