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Music is a major part of many people’s lives. But most of them believe that singing is a talent that you are born with, you cannot learn good singing through practice. We all have that inner voice, that sounds much better than our actual singing. And that voice may be the reason why you cannot sing on pitch!
This inner voice is known as subvocalization. According to ScienceDirect, it is the internal singing or speaking of lyrics or sentences, accompanied by subtle, often unconscious, movements of facial muscles and other body parts. A 2018 study published in the Psychophysiology revealed that imagining the act of singing before actually vocalizing improved pitch accuracy. On the other hand, physical preparation through body movements led to more pitch errors. Essentially, mental rehearsal helps, while physical tension hinders.
Another significant factor affecting our ability to sing in tune is tempo. Research published in The Journal of the Acoustical Society of America explored how singing speed impacts pitch. The study found that occasional singers matched professional singers' pitch accuracy at slower tempos. However, when singing faster, pitch errors increased significantly.
Beyond tempo, the ability to sing in tune hinges on three core elements which are pitch accuracy, rhythmic precision, and note memory. It's important to remember that even trained singers experience pitch imperfections. In fact, perfect pitch is exceedingly rare, occurring in only one to five out of 10,000 people, according to Scientific American.
Ever noticed a family trend of off-key singing? Genetics could be playing a role. Experts have long suspected a link between genetic predisposition and singing ability. According to a 2022 study published in the iScience journal, singing is a mix of what you're born with (your genes) and what you learn from your surroundings. To figure this out, scientists asked a large group of Australian twins to sing using a validated online singing tool. From these tests, they created a score that showed overall singing ability. They discovered that about 40% of how well someone sings comes from their genes. But, surprisingly, about 37% comes from their shared environment, like growing up in a musical home. This means that things like singing with your family as a kid or being around music early in life significantly affect how good you are at singing.
A 2012 study published in the NeuroImage has shown that when you listen to music, your brain's movement centers light up. Which means that there is more brain activity. But the more you practice, the less those centers light up. In the study researchers found that there was less activity in the brain when it knew the cords and was practicing for a while.
This means that practicing might help you control those little muscle movements that can throw your singing off. So, if you want to sing better, don't give up! Just keep practicing. Like music teachers always say, practice makes perfect, or at least closer to perfect. The more you sing, the better you'll get at controlling your muscles and staying in tune.
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For four years, 24-year-old Sneha suffered from severe, one-sided headaches that felt like a storm. These attacks, which were often accompanied by nausea and sensitivity to light, left her exhausted and anxious. Living in a small town, she saw many doctors, but nothing seemed to help. Her parents watched helplessly as their once cheerful daughter spent her days in a dark room, adding to Sneha's despair.
In a recent post on social media platform, Dr Sudhir Kumar MD, shared the case of a young women who was suffering with debilitating migraines. Despite the seriousness of her condition, doctors were unable to find what could help her. But a trick that she did out of sheer desperation, turned out to be the answer to her problem
One day, out of pure frustration, Sneha grabbed a cold pack, pressed it to her forehead, and wrapped a soft headband tightly around her head. To her surprise, the pain began to ease. The relief wasn't always complete, but even a little bit was enough to give her hope for the first time in years. This small "hack" made her feel like she wasn't completely helpless against her pain.
Also Read: Do This 1 Thing Before You Pop A Pill For Your Migraine, According To Pharmacist
When Sneha and her parents finally visited Dr. Kumar, they were tired but hopeful. She hesitantly told him about her simple trick, worried he might dismiss it. Instead, he reassured her that it was a safe method and explained that many patients find comfort from cold therapy and gentle pressure on the head. He told her she could continue to use it at home as a supportive measure.
After a careful evaluation, Dr. Kumar confirmed Sneha’s diagnosis of migraine. He started her on a proper medical treatment plan and, importantly, discussed preventive strategies. He advised her on simple lifestyle changes, such as getting consistent sleep, staying hydrated, managing stress, and avoiding certain food triggers that could set off her attacks.
As the doctor explained the plan, Sneha’s parents' faces filled with relief. They finally had answers and a clear path forward. Sneha herself smiled for the first time during their conversation, a genuine smile that showed she no longer felt alone in her fight against this debilitating illness.
Despite being a common problem, not many people know how to differentiate between a headache and migraine. According to the NKC Health organization, the key difference between a headache and a migraine is how severe the pain is. Headaches can range from a dull ache to a sharp pain, but migraines are often so debilitating that they can stop you in your tracks. A common way to remember migraine symptoms is the acronym POUND:
Dr Kumar highlighted several key points about migraines from Sneha’s experience.
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The recent outbreak of Ebola Virus Disease, with confirmed 81 cases have brought this question again to the world: Why are African countries disproportionately impacted by Ebola Outbreaks?
The Ebola Virus Disease (EVD) outbreaks have periodically occurred in affected regions of West and sub-Saharan Africa since the emergence of EVD in 1976. It continues to remain endemic in these regions to this day, whereas, it has been eradicated from other areas.
To understand why certain regions are disproportionately impacted, we must understand these followings.
Ebola’s incubation period can last from 2 to 21 days, but transmission begins only when symptoms appear. Once it strikes, the disease moves swiftly. Without supportive care, like IV fluids, antibiotics, dialysis, or specialized infection control, the fatality rate can soar up to 70% in places like Liberia, Sierra Leone, and Guinea. Compare that to the 2014 Ebola scare in the United States: nine cases, just one death, and zero spread beyond the initial outbreak. The contrast underscores a painful truth, Ebola is not only a medical problem but also a systemic one.
In countries like the US, hospitals are equipped with rehydration tools, multiple blood pressure medications, and well-trained doctors. They also have strict infection control systems to prevent the disease from moving beyond hospital walls.
In contrast, in many West African facilities, even the most basic supplies can run out. During the 2014 West Africa outbreak, some hospitals stocked just one type of blood pressure drug, hardly enough to save lives when patients’ bodies responded differently.
Also Read: Ebola Outbreak Update: Vaccines Continue To Arrive As Congo Yet Again Becomes Disease Hotspot
The disparity in staffing is equally grim. The US has about 245 doctors for every 100,000 people. Liberia, Sierra Leone, and Guinea? Just 1.4, 2.2, and 10 per 100,000, respectively, notes University of Michigan's School of Public Health. In a disease outbreak where immediate and skilled intervention can mean the difference between containment and chaos, this shortage is devastating.
But Ebola doesn’t just thrive on weak health systems, it feeds on broken trust. Many African nations hardest hit by Ebola have been scarred by years of civil conflict. Public health systems, roads, and communications were already fragile before the virus appeared. In Sierra Leone, Guinea, and Liberia, the memory of political violence and neglect fueled deep distrust of governments and outsiders.
During the 2014–2016 epidemic, attacks on doctors and aid workers became common. Rumors spread faster than the virus: some communities believed Ebola was a foreign invention, others feared that medical workers were spreading it deliberately. When burial traditions, like washing and touching the dead, were discouraged by officials, resistance turned violent, noted the report by University of Michigan. Cultural clashes and misinformation made every effort to contain the disease harder, often putting lives at even greater risk.
Ebola remains endemic in parts of Africa because it is more than a virus, it’s a reflection of the gaps in public health, governance, and community trust. The combination of limited resources, dangerously low numbers of medical staff, fragile infrastructures, and deep-seated distrust creates a cycle where each outbreak threatens to become a catastrophe.
Until these systems are rebuilt, hospitals resourced, communities engaged, and trust restored, Ebola will continue to lurk, waiting for its next chance to reemerge.
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Lyme disease is often described as a medical puzzle. Many patients expect recovery after treatment, but for some, lingering symptoms like fatigue, sweats, palpitations, or breathing difficulties, continue to disrupt daily life. Increasingly, researchers and practitioners are recognizing that a hidden co-infection may be responsible: Babesia.
Though transmitted by the same ticks that spread Lyme disease, Babesia is a parasite rather than a bacterium. This means that while standard antibiotics for Lyme may clear Borrelia burgdorferi (the Lyme-causing bacteria), they do not affect Babesia. If left untreated, the infection can stall or even derail recovery.
Alexis Chesney, MS, ND, LAc, who is also a naturopathic physician, acupuncturist, author, and educator specializing in Lyme and vector-borne disease, writes that Babesia is a malaria-like parasite that invades red blood cells, belonging to the phylum Apicomplexa.
Also Read: Ebola Outbreak Update: Vaccines Continue To Arrive As Congo Yet Again Becomes Disease Hotspot
First described in the late 19th century, it has since been identified in multiple species, with Babesia microti and Babesia duncani being most common in the U.S. The Centers for Disease Control and Prevention (CDC) recently reported a rise in cases across the Northeast and upper Midwest, even declaring Vermont, New Hampshire, and Maine as endemic states.
The parasite is transmitted by the blacklegged tick (Ixodes scapularis) in the East and Midwest, and Ixodes pacificus in the West. Transmission can occur within 36 hours of tick attachment, making timely removal critical.
Lyme disease is caused by bacteria, while Babesia is a protozoan parasite. The difference is crucial: antibiotics like doxycycline or ceftriaxone, typically prescribed for Lyme, have no effect on Babesia. When this parasitic infection is overlooked, patients often remain sick despite completing Lyme treatment.
This overlap of infections is common in regions where both pathogens circulate. Patients coinfected with Babesia and Lyme may experience a tougher, more prolonged illness, with night sweats, air hunger (difficulty breathing at rest), and palpitations serving as hallmark clues that Lyme alone isn’t to blame.
Babesia symptoms can vary widely, from mild to life-threatening. Common complaints include:
In severe cases, particularly in older adults, immunocompromised individuals, or those without a spleen, Babesia may cause hemolytic anemia, kidney problems, and organ stress.
Because symptoms overlap with Lyme disease, Babesia often goes undiagnosed without specific testing. Traditional options include:
Blood smear (Giemsa-stained): A classic but limited method, as only a tiny fraction of red blood cells may show parasites in early or chronic cases.
PCR testing: Detects Babesia DNA with high sensitivity in acute infections.
FISH (fluorescence in situ hybridization): A more advanced method that detects Babesia RNA, distinguishing between live and dead parasites and proving especially valuable in chronic disease.
Accurate testing is critical, as untreated Babesia can persist and block progress against Lyme.
Conventional treatment generally involves a combination of atovaquone and azithromycin for 7–10 days in mild to moderate cases. More severe or persistent infections may require longer regimens, sometimes with alternative drugs like clindamycin and quinine. However, treatment failures and drug resistance have been reported.
Naturopathic and integrative therapies are increasingly used alongside pharmaceuticals. Herbs such as Cryptolepis sanguinolenta, Artemisia annua (Sweet Annie), Polygonum cuspidatum (Japanese Knotweed), and Scutellaria baicalensis (Chinese skullcap) have shown anti-Babesia activity in laboratory studies. These botanicals not only target parasites but also support the immune system, reduce inflammation, and protect red blood cells.
Some practitioners also use anti-biofilm agents like serrapeptase or lumbrokinase to break down protective barriers that Babesia forms, making antimicrobial treatment more effective.
As Babesia spreads to new geographic regions, it is critical for healthcare providers to recognize its role in chronic illness. Ignoring Babesia may leave patients trapped in a cycle of incomplete Lyme recovery, chasing symptoms without lasting relief. A thorough approach that screens for co-infections and treats them comprehensively is essential.
For patients struggling with ongoing fatigue, sweats, or unexplained heart and lung symptoms after Lyme treatment, the missing piece could be Babesia. Recognizing and addressing this stealth parasite may be the key to finally breaking through to recovery.
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