You bend to lift a bag, sneeze a little too hard, or notice a strange bulge that was not there before. That small lump in your abdomen could be a hernia. While the word itself can sound alarming, hernia surgery today is routine and safe and comes in different types depending on your condition.

What’s a hernia?

A hernia is when an organ, often the gut or fatty tissue, pokes through a weak spot in your abdominal wall. Dr Pushkar Anand Singh, Senior Consultant – General and Laparoscopic Surgeon at Shri Ram Singh Hospital, explains it like this: “Think of it like a bulge in a worn-out tyre. The wall is supposed to hold everything in, but a weak spot lets the insides push out.”

This weak spot may form due to overexertion, like unsupervised weightlifting, constipation, or chronic cough, or it could be congenital (you were born with it). Sometimes it even develops at the site of an old surgery.

Who actually needs surgery?

Not everyone who discovers a hernia needs to be wheeled into an operating theatre right away. According to Dr Shrey Srivastava, Senior Consultant in Internal Medicine at Sharda Hospital, Greater Noida, surgery is usually advised when the hernia becomes painful, interferes with daily activities, or risks dangerous complications.

“Hernias that cannot be pushed back in, those that cause increasing pain, or worse, strangulated hernias where blood supply is cut off, need urgent surgery,” he says. Without timely intervention, this can lead to tissue death and potentially life-threatening infection.

On the flip side, if a hernia is tiny, painless, and not causing trouble, your doctor may simply keep an eye on it. But the general rule is that a hernia will not magically disappear on its own; sooner or later, surgery might be on the cards.

Open Hernia Repair

This is the old-school, tried-and-tested method. Surgeons make an incision right over the hernia, carefully push the bulging tissue back into place, repair the defect, and then reinforce the area with a mesh (like patching up that worn-out tyre).

“Open repair is highly cost-effective, and the outcomes are comparable with newer techniques,” says Dr Singh. For many patients, it remains the go-to option, especially when budget is a concern.

Laparoscopic Hernia Repair

Here, instead of one big cut, surgeons make three or four tiny incisions to slip in a camera and surgical instruments. The mesh is placed from the inside, which means no obvious scar at the hernia site.

The perks are smaller cuts, quicker healing, and less pain afterwards. However, it costs more, and as Dr Singh points out, “It is not recommended for patients with significant heart or lung issues, as the procedure puts more strain on the body.”

Robotic-Assisted Hernia Repair

Robots in surgery may sound like something out of a sci-fi movie, but they are here. Robotic-assisted hernia repair builds on the laparoscopic method, except the tools are controlled through a robotic console.

“The technology allows greater precision and 3D visualisation for the surgeon,” explains Dr Singh. It is slick and advanced, but it also comes with a hefty price tag. Since the outcomes are not dramatically better than laparoscopic surgery, most hospitals do not see it as a routine option.

How do doctors decide which is right?

With three different techniques on the table, how do surgeons pick? Dr Singh says the choice depends on several factors like patient health, cost considerations, size of the hernia, and the urgency of the situation.

Open surgery might suit a patient looking for a straightforward, affordable solution. Laparoscopic repair works well for those who want a quicker recovery and can afford the added cost. Robotic surgery, while cool, is usually reserved for centres with access to the technology and patients willing to pay extra for the latest option.

Do not fear the word “surgery”

While the thought of going under the knife can be nerve-wracking, hernia surgeries today are routine, safe, and highly successful. Most patients return to normal activities within weeks, and the mesh reinforcement greatly reduces the risk of recurrence.

“Hernias are common, but complications can be dangerous,” Dr Srivastava reminds. “If you are experiencing persistent pain, visible bulges, or swelling that would not go back inside, do not delay consulting a doctor.”

Huntington’s disease (HD) is a condition that is hereditary and causes nerve cells in specific parts of the brain to slowly deteriorate and die. The disorder affects regions responsible for controlled and intentional movements, as well as those tied to mood, thinking, and personality. People with HD often develop jerky, dance-like body movements known as chorea, along with unusual postures and emotional or cognitive changes. For instance, they may experience sudden, uncontrollable motions in their hands, feet, face, or torso. These movements tend to worsen when the person feels anxious or distracted, and as the disease advances, they become more frequent and harder to manage.

Is Huntington’s Disease Genetic?

According to the National Institute of Neurological Disorders, Huntington’s disease is passed down genetically from parent to child. It follows an autosomal dominant inheritance pattern, meaning that only one copy of the faulty gene is enough to cause the illness. If a parent carries the gene, every child has a 50 percent chance of inheriting it. As it is known to be dominant, just one altered gene from either parent can trigger the disease.

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The institute further explains that children who do not receive the mutated gene will never develop HD and cannot pass it on to their own children.

What Causes Huntington’s Disease?

Huntington’s disease stems from a mutation in the HTT gene. This defect creates an abnormal stretch in the DNA sequence, called an expanded CAG repeat. The mutation leads to the production of a faulty version of the huntingtin protein, which contains an excessively long polyglutamine chain.

Over time, this unstable protein builds up in the brain and damages nerve cells. As the mutation is inherited in an autosomal dominant pattern, one copy of the defective gene is enough to cause the disorder.

Huntington’s Disease Can Now Be Treated With Gene Therapy

A new gene therapy treatment has shown promise in slowing the course of Huntington’s disease, marking what could be the first meaningful advance against this inherited brain disorder. In a recent clinical trial involving 29 patients in the early stages of HD, those who received a single, high-dose infusion of the therapy directly into the brain experienced a 75 percent slower progression over three years compared with the control group.

The results, shared by the Amsterdam-based gene therapy company uniQure, were considered statistically significant across several clinical measures. Researchers also found lower levels of a harmful protein linked to brain cell damage in the spinal fluid of participants who received the treatment. Encouraged by these results, uniQure has announced plans to seek regulatory approval next year.

“This gene therapy represents a major step forward,” said Dr. Sandra Kostyk, a neurologist at Ohio State University Wexner Medical Center and one of the study’s investigators. “The data are encouraging.”

She added that while slowing the illness could mean more years of independence for patients, it is not a cure. Because of the small number of participants, more research and long-term follow-up are still needed.

Huntington’s Disease Symptoms You Need to Be Aware Of

People with Huntington’s disease often lose control over their voluntary movements, which can affect daily functioning more severely than the involuntary jerks caused by chorea. Difficulties with voluntary movement can make it harder to work, communicate, and maintain independence.

According to the National Health Service, early signs may include trouble focusing or organizing tasks, forgetfulness, irritability, impulsive behavior, and changes in mood such as depression or anxiety. Other symptoms include small, uncontrollable jerks or twitches, clumsiness, and problems managing muscles.

Interestingly, symptoms can appear at any age but most often begin in a person’s 30s or 40s. When it develops before the age of 20, it is called juvenile Huntington’s disease. In such early-onset cases, symptoms may differ and the illness usually progresses more rapidly.

Health experts are raising alarms over a disturbing new trend in which drug users exchange blood to experience a shared high. Known as “bluetoothing,” this new practice is driving a sharp increase in HIV infections in regions such as Fiji and South Africa. Doctors warn that the recent rise in reported cases may only reveal a fraction of the potential damage linked to this dangerous act, which is also called “hotspotting.” But what exactly is behind this risky phenomenon, and why is it spreading?

What Is Bluetoothing and How Is It Linked to Rising HIV Cases?

Bluetoothing is a hazardous street practice where a person injects a potent drug such as heroin or meth, then withdraws a small amount of their own blood, now mixed with the drug, into a syringe and injects it into someone else to share the high. This process can continue from one person to the next, often using the same needle.

According to Brian Zanoni, a professor at Emory University who has studied drug-injecting behavior in South Africa, “it’s a cheap way to get high, but it comes with serious consequences as you’re essentially getting two doses for the price of one.” However, experts note that the effectiveness of this method is uncertain. Some believe the secondary injection produces only a mild high, while others argue it is just a placebo affect.

Why Is Bluetoothing So Dangerous for Health?

The idea of passing around drug-laced blood is so shockingly unsafe that for years, health professionals questioned whether it actually happened. Yet even if practiced by a small number of people, it can rapidly spread bloodborne diseases like HIV and hepatitis, prompting calls for an urgent public health response.

In Fiji, authorities have identified bluetoothing as a key factor behind an alarming rise in HIV rates. According to UNAIDS data, new HIV infections there increased tenfold between 2014 and 2024, leading to an official outbreak declaration in January.

About half of newly diagnosed individuals receiving antiretroviral treatment in Fiji reported contracting HIV through needle sharing, though it remains unclear how many knowingly exchanged blood. Most of these new cases were among people aged 15 to 34.

How Dangerous Is HIV and What Are Its Stages?

Without treatment, HIV gradually weakens the immune system, making the body vulnerable to serious infections. Over time, untreated HIV can develop into acquired immunodeficiency syndrome (AIDS). The progression of HIV occurs in three key stages for people who are not on treatment:

Acute HIV Infection

This first stage usually appears two to four weeks after exposure. Many people experience flu-like symptoms such as fever, rash, and headache. During this phase, the virus multiplies quickly and attacks the immune system’s CD4 cells, leading to a very high viral load and increased risk of transmission.

Chronic HIV Infection

In this second stage, the virus continues to replicate at low levels. People often do not show symptoms, but without antiretroviral therapy (ART), this stage can advance to AIDS within a decade or sooner. With proper treatment, individuals can live in this stage for many years.

AIDS

The final and most severe stage occurs when the immune system is badly damaged. The body becomes unable to fight off infections and certain cancers. A diagnosis of AIDS is made when CD4 counts fall below 200 cells/mm³ or specific infections appear. At this stage, the viral load is high and transmission risk increases sharply. Without treatment, life expectancy is typically around three years.

Since “bluetoothing” is not a wireless or digital act but an extreme form of intravenous drug use, protection begins with avoiding environments where it might occur. The safest approach is never to start injecting drugs. Acknowledge that this practice is real and extremely dangerous, rather than dismissing it as an online rumor, especially in communities where drug abuse and poverty are common.

Scientists at Northwestern Medicine have made a major discovery: a much better and faster way to help bones heal. They wrote about their work in the science journal Nature Communications. We have always known that the loss of bones and teeth can be a permanent one, while we have artificial methods to fix the irregularities, we can no longer grow this tissue back.

However, this new method is very exciting because it could totally change how doctors create implants, the plates, screws, or replacement parts used in surgery. The main goal is to improve healing by getting the body to use its own natural tools to repair itself.

Can We Regrow Our Bones?

Guillermo Ameer, ScD, the lead researcher, believes this technique could revolutionize surgeries for bones and joints, (orthopedic) and for the face and skull (craniofacial). Instead of just being a passive structure, these new implants actively encourage healing using the body's own cells and repair mechanisms.

Dr. Ameer noted that damage from injuries is very common. Usually, doctors put in artificial materials like metal or plastic to fill the gap. He explained that their work, called regenerative medicine, is focused on helping the body regrow its own natural tissue to fix the damaged area permanently.

Can We Use Cells To Rebuild Bones?

Dr. Ameer’s team had previously developed a unique implant. Its surface isn't smooth; it has tiny, engineered micropillars (small bumps). When special repair cells called mesenchymal stem cells (MSCs) stick to the implant, these tiny bumps physically push on and change the shape of the cell's center, called the nucleus.

The big new finding from this latest study is that these cells—whose nuclei have been squished—start to release special healing proteins. These proteins actively promote bone growth in other nearby cells, not just the ones touching the implant.

How Can You Regrow Bones?

In their most recent experiment, the scientists watched closely to see exactly how the new implants caused bone to grow. They found that when the MSCs had their nuclei changed by the micropillars, they quickly increased their release of proteins that organize the extracellular matrix (ECM). Scaffolding is the process of construction where a temporary structure is made to support the workers while they do the construction. The ECM is basically the natural, supportive scaffolding around all tissues in the body

This newly organized scaffolding then tells other nearby MSCs to start making bone, even if they aren't directly on the implant. It's like a secret instruction being passed through the structural environment.

To test this in a real situation, the team placed the micropillar implants into mice with small holes in their skull bones. They saw that the cells on the implants made much more of a key protein called collagen, which is the main building block of bone structure. The result was significantly faster and better bone healing in the injured area.

What Is Cellular Communication?

These results show a special way cells talk to each other, called matricrine signaling. Instead of using direct contact or typical chemical messages, cells influence their neighbors by changing the extracellular matrix which is the scaffolding around them.

Dr. Ameer explained that when a cell's nucleus is deformed, its internal structure is rearranged. This makes the cell favor the production and release of proteins that tell other cells, "Start making bone!" He clarified that these released proteins actually change the environment (the matrix) surrounding nearby cells, instructing them to support new bone growth. This discovery opens up huge possibilities for designing implants that don't just act as supports, but actively guide and speed up the natural healing process.

Dr. Ameer also mentioned that this idea might be useful for repairing other tissues in the future, such as cartilage.

He stressed that losing cartilage, particularly in conditions like arthritis, is a major issue because the body has trouble regrowing it on its own. He noted that his team is already working on ways to use 3D printing to apply a similar strategy and help the body regenerate damaged cartilage.