Health and Me

Ishita Roy

Melanie Watson, Disability Advocate And Diff'rent Strokes Actress, Dies At 57

Melanie Watson, Diff'rent Strokes actress and disability advocate dies at 57 on Friday, December 26. The news was confirmed by her brother Robert, and told that she was hospitalized prior to her death. While her cause of death has not been revealed yet, her brother said that she was in hospital due to some bleeding issue.

<h2>What Is Osteogenesis Imperfecta?</h2>

Melanie was born with osteogenesis imperfecta or OI, which the Johns Hopkins Medicine notes is an inherited (genetic) bone disorder that is present at birth. It is also known as brittle bone disease. A child born with OI may have soft bones that break or fracture easily, bones that are not formed normally and other problems Signs and symptoms may range from mild to severe. 

OI also have eight different forms and the types vary greatly both within and between types. As per the Johns Hopkins Medicine, here are the eight types of OI:

Type I. Mildest and most common type. About 50% of all affected children have this type. There are few fractures and deformities

Type II. Most severe type. A baby has very short arms and legs, a small chest, and soft skull. He or she may be born with fractured bones. He or she may also have a low birth weight and lungs that are not well developed. A baby with type II OI usually dies within weeks of birth

Type III. Most severe type in babies who don’t die as newborns. At birth, a baby may have slightly shorter arms and legs than normal and arm, leg, and rib fractures. A baby may also have a larger than normal head, a triangle-shaped face, a deformed chest and spine, and breathing and swallowing problems. These symptoms are different in each baby.

Type IV. Symptoms are between mild and severe. A baby with type IV may be diagnosed at birth. He or she may not have any fractures until crawling or walking. The bones of the arms and legs may not be straight. He or she may not grow normally.

Type V. Similar to type IV. Symptoms may be medium to severe. It is common to have enlarged thickened areas (hypertrophic calluses) in the areas where large bones are fractured

Type VI. Very rare. Symptoms are medium. Similar to type IV.

Type VII. May be like type IV or type II. It is common to have shorter than normal height. Also common to have shorter than normal upper arm and thighbones.

Type VIII. Similar to types II and III. Very soft bones and severe growth problems.

This is a disease that could be passed on through genes and there are different types that could be passed on in different ways. These genes could be inherited from one or both parents, or could be passed on from an unexplained change or a mutation of the gene. 

Most babies who have OI have a defect of one of two genes. These genes help in collagen forming, which is a main part of connective tissue that connects and supports the whole body, including bones. Because of a certain defect, there is no enough collages, or the collagen is abnormal, which causes this disease. 

The main aim of treatment for this disease lies in prevention of deformities and fractures.

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Melanie Watson, Diff'rent Strokes actress and disability advocate dies at 57 on Friday, December 26. The news was confirmed by her brother Robert, and told that she was hospitalized prior to her death. While her cause of death has not been revealed yet, her brother said that she was in hospital due to some bleeding issue. What Is Osteogenesis Imperfecta?Melanie was born with osteogenesis imperfecta or OI, which the Johns Hopkins Medicine notes is an inherited (genetic) bone disorder that is present at birth. It is also known as brittle bone disease. A child born with OI may have soft bones that break or fracture easily, bones that are not formed normally and other problems Signs and symptoms may range from mild to severe. OI also have eight different forms and the types vary greatly both within and between types. As per the Johns Hopkins Medicine, here are the eight types of OI:Type I. Mildest and most common type. About 50% of all affected children have this type. There are few fractures and deformitiesType II. Most severe type. A baby has very short arms and legs, a small chest, and soft skull. He or she may be born with fractured bones. He or she may also have a low birth weight and lungs that are not well developed. A baby with type II OI usually dies within weeks of birthType III. Most severe type in babies who don’t die as newborns. At birth, a baby may have slightly shorter arms and legs than normal and arm, leg, and rib fractures. A baby may also have a larger than normal head, a triangle-shaped face, a deformed chest and spine, and breathing and swallowing problems. These symptoms are different in each baby.Type IV. Symptoms are between mild and severe. A baby with type IV may be diagnosed at birth. He or she may not have any fractures until crawling or walking. The bones of the arms and legs may not be straight. He or she may not grow normally.Type V. Similar to type IV. Symptoms may be medium to severe. It is common to have enlarged thickened areas (hypertrophic calluses) in the areas where large bones are fracturedType VI. Very rare. Symptoms are medium. Similar to type IV.Type VII. May be like type IV or type II. It is common to have shorter than normal height. Also common to have shorter than normal upper arm and thighbones. Type VIII. Similar to types II and III. Very soft bones and severe growth problems.What Causes OI?This is a disease that could be passed on through genes and there are different types that could be passed on in different ways. These genes could be inherited from one or both parents, or could be passed on from an unexplained change or a mutation of the gene. Most babies who have OI have a defect of one of two genes. These genes help in collagen forming, which is a main part of connective tissue that connects and supports the whole body, including bones. Because of a certain defect, there is no enough collages, or the collagen is abnormal, which causes this disease. The main aim of treatment for this disease lies in prevention of deformities and fractures.