Image Credit: Canva
Magnesium, sometimes called the "miracle mineral," is essential for more than 300 biochemical reactions in the body. Many women are not getting enough of this important nutrient. Research suggests that nearly 50% of the U.S. population may be magnesium-deficient, which can significantly impact overall health, energy levels, and disease risk.
Magnesium, from the functioning of the muscles and smooth transmission of the nerves to energy production in the form of ATP and cardiovascular health, keeps the body in balance. Why, then, is this deficiency so widespread, and why do women need to pay that much attention to their magnesium intake?
The recommended daily intake of magnesium is 320 mg for women, but most women take in much less than this amount through their diet. Magnesium is particularly important for women because of its effects on hormonal balance, metabolic health, and bone strength. Women experience a range of physiological changes throughout their lives-from puberty to pregnancy to menopause-and each of these physiological changes increases the body's magnesium requirements.
A few health conditions unique to women will benefit from an increase in magnesium intake, including:
Polycystic ovarian syndrome is often associated with insulin resistance in women, leading to weight gain and metabolic disorders. Magnesium has been found to improve the body's sensitivity to insulin and, therefore, control blood sugar levels, making it an important nutrient for women who suffer from PCOS.
Low magnesium levels have been associated with higher anxiety, irritability, and depression. Women who have PMS often suffer from mood swings and discomfort; magnesium may be helpful in relieving such symptoms. According to studies, supplementing with magnesium and vitamin B6 has greatly reduced the symptoms of PMS.
The transition towards menopause means that the level of magnesium decreases, worsening the symptoms that characterize it hot flashes and mood swings among others. Supplementing with magnesium has greatly been proven to lighten such symptoms and, in turn, improve the overall well-being of women.
Magnesium is essential for the pregnant woman to support the fetal development process, preventing complications like high blood pressure, preeclampsia, and preterm birth. Many prenatal vitamins contain magnesium; however, food sources should not be ignored.
They have a greater susceptibility to osteoporosis post-menopause. Magnesium has been proven crucial for bone-building activity and bone density, making bones less likely to break or get osteoporosis. The person cannot assimilate calcium unless enough magnesium is provided; then they develop brittle bones.
Women are also the most affected in chronic fatigue syndrome and fibromyalgia, which have been associated with low magnesium levels. Since magnesium is an important player in cellular energy production, supplementation can combat fatigue and increase general well-being.
Women are three times more likely than men to suffer from autoimmune diseases, most of which are inflammation-based. Magnesium helps modulate immune responses and reduce oxidative stress, which would decrease the incidence of flare-ups in autoimmune conditions.
Cardiovascular diseases are one of the leading causes of death for women, while magnesium is critical in maintaining regular heart rhythms and preventing blood clotting, also regulating blood pressure. A magnesium-rich diet could help reduce risk factors for hypertension and heart diseases.
Migraines can be made worse by the low level of magnesium. It assists in reducing blood vessel contraction, hence making one experience migraines less often and severe less.
It is considered as an invisible deficiency since symptoms tend to appear more subtly, sometimes easily neglected. Common symptoms of this are;
If you are experiencing several of these symptoms, then it may be time to reassess your magnesium intake and look at topping them up with diet or supplementation.
The best way to ensure adequate magnesium levels is through a balanced diet rich in magnesium-containing foods. Some of the top dietary sources include:
For those who don't get an adequate amount through food alone, high-quality supplemental magnesium in either glycinate or citrate helps fill the void. Supplementation should be exercised with caution since excessive intake must be avoided, though.
While magnesium is important, excessive amounts can cause side effects like diarrhea, nausea, muscle weakness, and low blood pressure. The upper limit for magnesium supplements is about 350 mg per day, so it is essential to adhere to the recommended doses. Women with kidney problems should consult their doctor before taking magnesium supplements because excess magnesium can accumulate in the body if kidney function is impaired.
Magnesium is one of those minerals that so often gets left out, and yet it helps with so much in the body of a female. It impacts hormonal balance to heart health to stress relief: magnesium is pivotal to overall health. If you think you're deficient, be sure to push magnesium-rich foods or talk with your healthcare professional about supplementing.
Subclinical magnesium deficiency: a principal driver of cardiovascular disease and a public health crisis. Open Heart. 2018
The Importance of Magnesium in Clinical Healthcare. Scientifica (Cairo). 2017
Credits: Canva
For many, beer is more than just a drink—it’s part of social culture, celebrations, and downtime rituals. But new research suggests there may be something more unsettling hiding in your pint glass, toxic “forever chemicals.” A recent study published in ACS Environmental Science & Technology has revealed that over 95 percent of beers tested across the United States and abroad contain traces of per- and polyfluoroalkyl substances (PFAS).
PFAS are a large family of manmade chemicals developed in the mid-20th century for their resistance to water, oil, heat, and stains. They’re found in non-stick cookware, water-resistant clothing, food packaging, firefighting foams, and even cosmetics. Their industrial versatility, however, comes at a steep cost: PFAS do not naturally break down, earning them the nickname “forever chemicals.”
Mounting evidence links PFAS exposure to health problems, including hormonal disruption, weakened immunity, high cholesterol, liver damage, fertility challenges, and increased risks of certain cancers. The U.S. Environmental Protection Agency (EPA) recently set strict new limits on PFAS in drinking water, underscoring the urgency of the problem.
Beer is made primarily of water, and researchers suspected that if municipal water supplies were contaminated with PFAS, those chemicals might carry over into the brewing process. That’s exactly what this new study confirmed.
The research team, led by Jennifer Hoponick Redmon, adapted an EPA-approved method normally used for testing drinking water and applied it to beer. They examined 23 beers from across the U.S. including those brewed in North Carolina, Michigan, Colorado, California, Massachusetts, Pennsylvania, Wisconsin, and Missouri as well as beers imported from Mexico and the Netherlands.
The findings were striking: PFAS were present in 95 percent of samples, with contamination levels strongly correlating to the quality of local water supplies. Beers brewed near the Cape Fear River Basin in North Carolina, a region notorious for PFAS pollution, contained the highest concentrations and the widest mix of chemicals.
This is the first study to directly link municipal water contamination to PFAS levels in U.S. retail beer. While breweries often use filtration systems, these methods are not designed to capture PFAS. As a result, the contamination that begins in local rivers and reservoirs can persist all the way into the finished product.
The results highlight an uncomfortable reality: PFAS contamination doesn’t just stay in the water—it spreads into food and beverages consumed daily. Beer is only the latest product to show evidence of contamination, joining an already long list that includes bottled water, fish, dairy, and vegetables.
“As an occasional beer drinker myself, I wondered whether PFAS in water supplies was making its way into our pints,” said Redmon in a statement. “I hope these findings inspire water treatment strategies and policies that help reduce the likelihood of PFAS in future pours.”
Her comments echo a growing consensus among environmental health experts that solving PFAS contamination requires systemic change. Since individual breweries can do little to alter the chemical composition of municipal water, solutions must come from upgrading public water systems and enforcing stricter industrial discharge regulations.
For consumers, the immediate concern is how much PFAS exposure from beer actually contributes to long-term health risks. The EPA’s new standards set the safe threshold for certain PFAS, including perfluorooctanesulfonate (PFOS) and perfluorooctanoic acid (PFOA), at near-zero levels in drinking water. While the amounts found in beer are relatively small per serving, scientists warn that cumulative exposure across multiple sources—water, food, packaging, and now beer—poses a real risk over time.
The issue extends far beyond breweries. PFAS contamination has been detected in rivers, groundwater, and public water systems across the United States and around the globe. A seven-fold increase in Google searches for “PFAS” in the past five years reflects rising public concern as awareness grows.
Everyday products contribute to the exposure cycle: waterproof clothing, stain-resistant upholstery, non-stick pans, and even cosmetics like waterproof mascaras can shed PFAS into the environment. Once in the ecosystem, they seep into soil, water, and eventually our bodies.
For casual drinkers, a pint of beer is unlikely to be the sole driver of PFAS-related health risks. However, the study reinforces a broader message: PFAS are everywhere, and beer is another reminder of how pervasive they’ve become. Health experts emphasize that reducing exposure across all possible sources is key, since the chemicals accumulate in the body over time.
As Dr. Redmon’s team points out, the solution doesn’t lie in avoiding beer altogether—it lies in addressing the root of the problem: contaminated water systems and unchecked chemical pollution.
Credits: Health and me
Heavy drinking is a recognized risk factor for liver injury, but a new report by the University of California San Diego reveals an previously unrecognized mechanism that amplifies this damage. Long-term drinking not only causes direct damage to the liver but also initiates a toxic cycle involving the gut, in which bacteria spill into the blood and attack the liver, leading to cumulative injury.
This new research, published in Nature, combines human liver biopsies and mouse models of alcohol-associated liver disease to reveal a molecular pathway that could redefine our understanding of alcohol-related liver damage.
The research points out a protein known as mAChR4, which is present in the small intestine, and is critical in supporting the immune defenses of the gut. Prolonged alcohol consumption inhibits the synthesis of mAChR4 and hence the creation of GAPs, or goblet cell-associated antigen passages. GAPs are the unique structures that facilitate the education of the immune system in order to effectively respond to microbes.
When GAPs do not form, gut bacteria have a greater likelihood of leaking from the intestines and infecting organs such as the liver. Inside the liver, the bacteria have the ability to increase inflammation and tissue damage, adding to the liver damage that has been inflicted by alcohol. In essence, alcohol compromises the gut-liver barrier while, at the same time, establishing an environment in which bacterial invasion can occur, making it a self-sustaining cycle of harm.
The UC San Diego researchers determined that restoring mAChR4 function had the potential to fix GAPs and strengthen the immune system's attack on rogue gut bacteria. This therapy, which may be accomplished with drugs that directly stimulate mAChR4 or by altering related signaling pathways, decreased liver damage in laboratory models.
Although limiting alcohol intake is the most straightforward method of safeguarding the liver, targeting mAChR4 represents a promising therapeutic strategy for alcohol abusers or individuals already damaged by liver injury. Curiously, mAChR4 is also expressed in the brain, where it modulates habit formation and addiction. Reduced levels of this protein have been noted in individuals with alcohol use disorder, paving the way for future treatments that target both liver function and addictive behavior.
Outside the liver, alcohol can significantly interfere with gut health in several ways. Six mechanisms explain how long-term drinking damages the digestive tract:
Alcohol loosens the lower esophageal sphincter, permitting stomach acid to flow back and produce heartburn or gastroesophageal reflux disease (GERD).
Excessive drinking upsets the balance of bacteria in the intestines, favoring "bad" bacteria that stimulate inflammation and intestinal permeability. This causes a leaky gut syndrome, where poisons enter the blood.
Alcohol interferes with mucus production within the stomach, which makes the lining susceptible to inflammation, pain, nausea, and vomiting.
Alcohol changes sugar metabolism and microbial equilibrium, in addition to stimulating yeast overgrowth, all of which promote gas production and bloating.
Long-term drinking results in alcoholic fatty liver disease and alcoholic steatohepatitis. Fat deposition, inflammation, and toxin-induced cell damage can evolve into cirrhosis, liver cancer, or liver failure, sometimes requiring transplantation.
Alcohol is metabolized by the pancreas into poisonous byproducts that can inflame the organ in an illness known as pancreatitis. Regular heavy alcohol consumption, particularly along with smoking, increases the risk for acute and chronic pancreatitis and long-term complications such as diabetes and pancreatic carcinoma.
The recognition of mAChR4 as a central figure in the gut-liver axis reveals a number of avenues for research. Drug therapies that enhance mAChR4 function within the gut would restore GAP function, reinforcing immunity against translocating bacteria. At the same time, drugs that increase mAChR4 function in the brain would decrease alcohol craving and treat the disorder itself, attacking both cause and effect of chronic alcoholism.
This dual potential makes mAChR4 an especially attractive target in the fight against alcohol-related disease. Clinical trials of mAChR4-targeted drugs are already in progress for other neurological disorders like schizophrenia, which provides the hope that those drugs might one day find use against alcohol-related liver disease as well.
Appreciation of gut-liver interaction puts the wider public health significance of chronic alcohol use into perspective. Interventions to address gut integrity, immune response, and liver defense may be able to complement traditional measures aimed at decreased alcohol consumption.
In addition, increased awareness of how alcohol impacts not only the liver but also gut bacteria may encourage behavior change. Most individuals downplay the insidious, long-term effects of alcohol on digestive health, thinking damage to the liver only follows decades of misuse. Changes in gut barrier function and bacterial translocation, however, can hasten liver injury even in moderate chronic drinkers.
For individuals, the clearest path to protecting liver health remains reducing alcohol intake. However, for those who struggle with dependency, therapies targeting mAChR4 may provide an adjunctive strategy to prevent liver injury while supporting recovery.
Nutrition and lifestyle also contribute to sustaining liver and gut health. A diet high in fiber, probiotics, and anti-inflammatory food can promote healthy gut bacteria and immune function, possibly preventing some of the adverse effects of alcohol.
The UC San Diego research reveals a key feedback loop of alcohol-induced liver damage: long-term drinking compromises gut barriers, enabling bacteria to travel to the liver, exacerbating injury. Targeting mAChR4 might interrupt this self-destructive process, providing new hope for treating alcohol-related liver disease.
This study supports an important message: alcohol not only harms the liver outright; it upsets the gut-liver axis and compounds damage. With advancing science, learning more about the molecular mechanisms underlying these effects can inform preventive measures and novel treatments and offer hope for millions globally affected by alcohol-associated liver disease.
(Credit-Canva)
A common phrase that many people say things like ‘I feel so bloated’ or ‘My stomach looks so bloated’ but what does it exactly mean? When a person feels bloated it is more than just an aesthetic or look issue. It's not just the fact that their abdomen looks swollen, but it means that they are feeling lot of pressure in their stomach due to build up of gas.
It is an uncomfortable sensation that can distract you from your work, make it difficult to focus on important things as well as causing you abdominal pain.
Considering this, and it being a common issue for many people, you will find a barrage of online advice telling you to drink ‘anti-bloating’ drink or try ‘that anti-bloating’ exercise. To deal with this, however, one must know exactly why they are bloated in the first place.
Dr. Saurabh Sethi, a gastroenterologist, addresses a very common concern in his practice: chronic bloating. He explains that if you feel bloated every time you eat, it's likely due to one of three reasons he frequently sees in his patients.
While many people believe they know the cause, Dr. Sethi points out that the most common culprit is often overlooked. He aims to provide clarity on these issues, noting that the final reason he discusses is the one most likely causing problems for the average person. By understanding the root cause, individuals can take the right steps toward finding relief.
Dr. Sethi explains that a common cause of bloating is constipation, and it isn't always what you think. While many people believe they're fine as long as they have a daily bowel movement, the real issue can be a "slow colon." This means food waste moves through your digestive system at a sluggish pace.
When this happens, food sits in your intestines for a much longer time than it should. This creates a perfect environment for the bacteria in your gut to break down the leftover food through a process called fermentation. This process releases a significant amount of gas, leading to that uncomfortable feeling of fullness, pressure, and bloating you experience after eating.
In this case, the main problem is an overly sensitive gut. Dr. Sethi points out that for people with Irritable Bowel Syndrome (IBS) or functional dyspepsia, the nerves and muscles in the intestines can move in a chaotic or irregular way. Instead of a smooth, coordinated process, the muscles can spasm or move too quickly or too slowly.
This irregular motion can easily trap pockets of gas, causing pressure and a visibly distended abdomen. This gut dysfunction is often linked to an imbalance in the gut's bacterial environment, which can make the intestines more sensitive to normal digestive processes and cause them to overreact, leading to heightened symptoms of gas and bloating.
According to Dr. Sethi, your diet is the most frequent and significant cause of chronic bloating. The primary culprits are specific types of carbohydrates that your small intestine has a hard time fully absorbing. These include lactose found in dairy products, fructose from many fruits and sweeteners like high-fructose corn syrup, and fructans in common foods like wheat, onions, and garlic.
When these undigested carbs reach your large intestine, the bacteria there have a feast, releasing a huge amount of gas as a byproduct. Dr. Sethi emphasizes that even healthy foods, such as broccoli, beans, and certain fruits, can trigger these symptoms if your digestive system is sensitive to these particular ingredients.
© 2024 Bennett, Coleman & Company Limited