Health and Me

Riya Baibhawi

Move over white noise—pink noise is the latest trend gaining popularity among people seeking better <span>sleep</span> and deeper relaxation. This softer, more soothing sound is fast becoming a go-to tool, alongside brown and green noise, in what some call a 'rainbow of relaxation.' Each of these coloured sounds is believed to influence sleep, focus, and overall calm—but is there any science to support the claims?

<h2>What Is <span rel="noindex">Pink Noise</span>?</h2>

Though the research is still in its early stages, that hasn’t stopped people from spending hours listening to these sounds on YouTube or through meditation apps that now offer curated colour-noise playlists under paid subscriptions.

To understand pink noise, it's helpful to first look at <span>white noise</span> — the most well-known of the group. White noise resembles the static heard on a television or radio and is defined by sound engineers as containing equal volume across all frequencies detectable by the human ear. It gets its name from white light, which contains all visible colour wavelengths.

However, white noise’s high frequencies can be harsh. That’s where pink noise comes in — it reduces the intensity of higher frequencies, producing a softer, lower-pitched sound similar to rain or ocean waves. Brown noise goes even deeper, offering a richer rumble that many find soothing.

While pink, white, and brown noise have clear definitions in audio engineering, other colour noises—like green or blue—are newer and more loosely defined.

Though still limited, there’s some emerging science behind colour noises. A recent review of small studies found that both white and pink noise may offer minor benefits to people with attention-deficit/hyperactivity disorder (ADHD). According to Joel Nigg, an ADHD researcher at Oregon Health &amp; Science University, these noises may stimulate the brain without offering actual information—which helps prevent distraction.

White noise has also been used in tinnitus treatment—helping mask the ringing or buzzing in the ears. Meanwhile, scientists at Northwestern University are exploring how short bursts of pink noise might boost slow-wave brain activity during deep sleep. Preliminary studies suggest that these pink noise pulses could enhance memory and promote relaxation.

Dr. Roneil Malkani, a neurology professor at Northwestern University Feinberg School of Medicine, explains that pink noise closely mirrors the natural frequency distribution of brain waves seen during slow-wave sleep. This similarity could help explain its calming effect. If the research holds up, it may lead to devices that use personalised pink noise to improve memory or sleep quality. However, Dr. Malkani cautions that “there’s still a lot of work we have to do.”

If colour noises feel relaxing or help block distractions, they can be a helpful tool. Just be mindful of volume. “Keep it quiet to avoid hearing damage and give your ears regular breaks,” Nigg advises. As the trend grows, pink noise may prove to be more than just a fad, offering a gentle, non-invasive way to improve rest and well-being.

<strong>ALSO READ:</strong> <a href="https://www.healthandme.com/health-wellness/what-is-moon-breathing-how-does-it-promote-sleep-article-151100992" data-type="tilCustomLink">What Is Moon Breathing? How Does It Promote Sleep?</a>

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Is Collagen Worsening Type 2 Diabetes?

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Move over white noise—pink noise is the latest trend gaining popularity among people seeking better sleep and deeper relaxation. This softer, more soothing sound is fast becoming a go-to tool, alongside brown and green noise, in what some call a 'rainbow of relaxation.' Each of these coloured sounds is believed to influence sleep, focus, and overall calm—but is there any science to support the claims?What Is Pink Noise?Though the research is still in its early stages, that hasn’t stopped people from spending hours listening to these sounds on YouTube or through meditation apps that now offer curated colour-noise playlists under paid subscriptions.To understand pink noise, it's helpful to first look at white noise — the most well-known of the group. White noise resembles the static heard on a television or radio and is defined by sound engineers as containing equal volume across all frequencies detectable by the human ear. It gets its name from white light, which contains all visible colour wavelengths.However, white noise’s high frequencies can be harsh. That’s where pink noise comes in — it reduces the intensity of higher frequencies, producing a softer, lower-pitched sound similar to rain or ocean waves. Brown noise goes even deeper, offering a richer rumble that many find soothing.While pink, white, and brown noise have clear definitions in audio engineering, other colour noises—like green or blue—are newer and more loosely defined.What Does Research Say?Though still limited, there’s some emerging science behind colour noises. A recent review of small studies found that both white and pink noise may offer minor benefits to people with attention-deficit/hyperactivity disorder (ADHD). According to Joel Nigg, an ADHD researcher at Oregon Health & Science University, these noises may stimulate the brain without offering actual information—which helps prevent distraction.White noise has also been used in tinnitus treatment—helping mask the ringing or buzzing in the ears. Meanwhile, scientists at Northwestern University are exploring how short bursts of pink noise might boost slow-wave brain activity during deep sleep. Preliminary studies suggest that these pink noise pulses could enhance memory and promote relaxation.Dr. Roneil Malkani, a neurology professor at Northwestern University Feinberg School of Medicine, explains that pink noise closely mirrors the natural frequency distribution of brain waves seen during slow-wave sleep. This similarity could help explain its calming effect. If the research holds up, it may lead to devices that use personalised pink noise to improve memory or sleep quality. However, Dr. Malkani cautions that “there’s still a lot of work we have to do.”Is It Safe to Try?If colour noises feel relaxing or help block distractions, they can be a helpful tool. Just be mindful of volume. “Keep it quiet to avoid hearing damage and give your ears regular breaks,” Nigg advises. As the trend grows, pink noise may prove to be more than just a fad, offering a gentle, non-invasive way to improve rest and well-being.ALSO READ: What Is Moon Breathing? How Does It Promote Sleep?

Pink Noise Can Help You Sleep Better—Know Everything About It

pink noise can help you sleep better—know everything about it

Ishita Roy

Health News

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One Crucial Habit Every Diabetic Is Forgetting – And It Starts With Your Toothbrush

one crucial habit every diabetic is forgetting – and it starts with your toothbrush

A new study published in the Journal of the American Chemical Society offers critical insight into the biological mechanisms underlying <span>type 2 diabetes</span>. Researchers from the Indian Institute of Technology Bombay (IIT Bombay), in collaboration with IIT Kanpur and the Chittaranjan National Cancer Institute (CNCI), Kolkata, have identified a key trigger that accelerates the progression of this widespread disease: the structural protein <span>collagen</span> I.

Type 2 <span>diabetes</span> currently affects over 500 million people worldwide, and numbers are expected to rise sharply in the coming decades. The disease is primarily driven by a combination of genetics, lifestyle factors, and complex cellular mechanisms. At its core lies the dysfunction of pancreatic β-cells, the insulin-producing cells responsible for regulating blood sugar levels.

As diabetes develops, β-cells either fail to produce enough insulin or the body’s cells become resistant to it. A lesser-known yet crucial hormone, amylin, is also secreted by these β-cells and plays a vital role in managing blood sugar after meals. However, in diabetic conditions, excessive amylin production leads to misfolding and toxic clumping, which damages β-cells and accelerates disease progression.

<h4>Collagen I Accelerates Amylin Clumping</h4>

In the latest study, the research team pinpointed fibrillar collagen I, a common component of the extracellular matrix, as a key factor driving the toxic aggregation of amylin. Found abundantly in connective tissues like skin and bones, collagen I is also present in the pancreatic environment—particularly in diabetic tissues where it is elevated.

“Every tissue is composed of cells and an extracellular matrix that provides structural support. In diabetic pancreatic tissue, this matrix, especially collagen I, becomes more prominent,” explained Prof. Shamik Sen, the study’s lead investigator from the Department of Biosciences and Bioengineering at IIT Bombay.

The researchers discovered that collagen I acts like a scaffold or platform, accelerating the misfolding and aggregation of amylin, which in turn damages β-cells. This discovery adds a new layer to understanding why the disease worsens over time, even with treatments targeting cellular pathways.

<h4>Biophysical Evidence Supports Findings</h4>

To investigate how collagen I interacts with amylin, the team used a suite of advanced biophysical tools. These included surface plasmon resonance to measure binding strength, atomic force microscopy to study molecular adhesion, thioflavin T fluorescence to track aggregation speed, and NMR spectroscopy to identify interacting regions of the molecules.

“Amylin almost coats the collagen fibres, forming stable, toxic aggregates that cells struggle to clear,” said Prof. Sen. The behavior of amylin on collagen fibres resembled trains moving on tracks—quickly and with destructive momentum.

Computer simulations by Prof. Prasenjit Bhaumik’s group at IIT Bombay confirmed that fibrillar collagen I accelerates the toxic aggregation process, offering further validation of the molecular interaction.

<h4>Biological Evidence from Mouse and Human Tissues</h4>

The team extended their study to biological samples from diabetic mice and humans. With the help of Prof. Hamim Zafar and Prof. Sai Prasad Pydi from IIT Kanpur, and Dr. Sankhadeep Dutta from CNCI, they analyzed single-cell data and tissue architecture.

The findings were striking: as diabetes progressed, both collagen and amylin levels rose, accompanied by damage to pancreatic islets—clusters of cells that house insulin-producing β-cells.

<h4>Testing the Combined Effect on Cells</h4>

To test the functional impact, the researchers grew lab-engineered β-cells on collagen gels containing amylin. These cells showed increased oxidative stress, reduced insulin production, and higher rates of cell death, compared to controls grown without collagen or amylin.

This suggests that the extracellular environment, particularly collagen I, plays a central role in worsening β-cell dysfunction in diabetes.

The findings could explain why many diabetes treatments fall short—they overlook the external microenvironment contributing to disease progression. “Unless we disrupt the interaction between amylin and collagen, we may not be able to eliminate the toxic pancreatic environment,” said Prof. Sen.

Looking ahead, the team is working on cryo-electron microscopy (cryo-EM) models to visualize how amylin and collagen interact at the molecular level. They are also exploring 3D tissue engineering strategies to restore pancreatic function by replicating healthy extracellular conditions.

type 2 diabetes

health and wellness

diabetes

collagen

collagen and diabetes

A new study published in the Journal of the American Chemical Society offers critical insight into the biological mechanisms underlying type 2 diabetes. Researchers from the Indian Institute of Technology Bombay (IIT Bombay), in collaboration with IIT Kanpur and the Chittaranjan National Cancer Institute (CNCI), Kolkata, have identified a key trigger that accelerates the progression of this widespread disease: the structural protein collagen I.A Rising Global Health CrisisType 2 diabetes currently affects over 500 million people worldwide, and numbers are expected to rise sharply in the coming decades. The disease is primarily driven by a combination of genetics, lifestyle factors, and complex cellular mechanisms. At its core lies the dysfunction of pancreatic β-cells, the insulin-producing cells responsible for regulating blood sugar levels.As diabetes develops, β-cells either fail to produce enough insulin or the body’s cells become resistant to it. A lesser-known yet crucial hormone, amylin, is also secreted by these β-cells and plays a vital role in managing blood sugar after meals. However, in diabetic conditions, excessive amylin production leads to misfolding and toxic clumping, which damages β-cells and accelerates disease progression.Collagen I Accelerates Amylin ClumpingIn the latest study, the research team pinpointed fibrillar collagen I, a common component of the extracellular matrix, as a key factor driving the toxic aggregation of amylin. Found abundantly in connective tissues like skin and bones, collagen I is also present in the pancreatic environment—particularly in diabetic tissues where it is elevated.“Every tissue is composed of cells and an extracellular matrix that provides structural support. In diabetic pancreatic tissue, this matrix, especially collagen I, becomes more prominent,” explained Prof. Shamik Sen, the study’s lead investigator from the Department of Biosciences and Bioengineering at IIT Bombay.The researchers discovered that collagen I acts like a scaffold or platform, accelerating the misfolding and aggregation of amylin, which in turn damages β-cells. This discovery adds a new layer to understanding why the disease worsens over time, even with treatments targeting cellular pathways.Biophysical Evidence Supports FindingsTo investigate how collagen I interacts with amylin, the team used a suite of advanced biophysical tools. These included surface plasmon resonance to measure binding strength, atomic force microscopy to study molecular adhesion, thioflavin T fluorescence to track aggregation speed, and NMR spectroscopy to identify interacting regions of the molecules.“Amylin almost coats the collagen fibres, forming stable, toxic aggregates that cells struggle to clear,” said Prof. Sen. The behavior of amylin on collagen fibres resembled trains moving on tracks—quickly and with destructive momentum.Computer simulations by Prof. Prasenjit Bhaumik’s group at IIT Bombay confirmed that fibrillar collagen I accelerates the toxic aggregation process, offering further validation of the molecular interaction.Biological Evidence from Mouse and Human TissuesThe team extended their study to biological samples from diabetic mice and humans. With the help of Prof. Hamim Zafar and Prof. Sai Prasad Pydi from IIT Kanpur, and Dr. Sankhadeep Dutta from CNCI, they analyzed single-cell data and tissue architecture.The findings were striking: as diabetes progressed, both collagen and amylin levels rose, accompanied by damage to pancreatic islets—clusters of cells that house insulin-producing β-cells.Testing the Combined Effect on CellsTo test the functional impact, the researchers grew lab-engineered β-cells on collagen gels containing amylin. These cells showed increased oxidative stress, reduced insulin production, and higher rates of cell death, compared to controls grown without collagen or amylin.This suggests that the extracellular environment, particularly collagen I, plays a central role in worsening β-cell dysfunction in diabetes.The findings could explain why many diabetes treatments fall short—they overlook the external microenvironment contributing to disease progression. “Unless we disrupt the interaction between amylin and collagen, we may not be able to eliminate the toxic pancreatic environment,” said Prof. Sen.Looking ahead, the team is working on cryo-electron microscopy (cryo-EM) models to visualize how amylin and collagen interact at the molecular level. They are also exploring 3D tissue engineering strategies to restore pancreatic function by replicating healthy extracellular conditions.

is collagen worsening type 2 diabetes?

Tanya Dutt

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Allergic Reactions Lead To Unnecessary Hospitalizations In Kids – How Can Parents Help Prevent This?

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As wildfires become an all-too-familiar headline across the globe, their visible devastation—scorched forests, lost homes, and displaced communities—often overshadows a more insidious threat: the impact of wildfire smoke on human health. While the immediate dangers of flames and evacuations are clear, a new study published in Nature Medicine reveals that wildfire smoke does far more than irritate eyes and throats. It can fundamentally alter the immune system, potentially making people more susceptible to illness long after the smoke has cleared.

The study, led by researchers from the Harvard T.H. Chan School of Public Health, found that people exposed to wildfire smoke exhibit changes in gene expression and immune function, even if they’re otherwise healthy. This suggests that the health consequences of wildfires may extend far beyond the immediate coughing, wheezing, and watery eyes that so many experience during fire season.

The research, led by Dr. Kari Nadeau of the Harvard T.H. Chan School of Public Health, provides the most detailed look yet at how wildfire smoke affects the body on a cellular level. By analyzing blood samples from 31 firefighters and civilians exposed to wildfire smoke and comparing them to 29 unexposed individuals, the study uncovered a complex web of immune changes.

People exposed to wildfire smoke showed a marked increase in memory immune cells—cells that typically provide long-term immunity. At first glance, this might seem beneficial, but the study also found elevated biomarkers of inflammation and immune activity, as well as changes in dozens of genes linked to allergies and asthma. In short, the immune system was not just activated—it was thrown into overdrive, potentially increasing vulnerability to illness.

Dr. Mary Johnson, the study’s lead researcher, explains, “Our findings demonstrate that the immune system is extremely sensitive to environmental exposures like fire smoke, even in healthy individuals. Knowing exactly how may help us detect immune dysfunction from smoke exposure earlier and could pave the way for new therapeutics to mitigate, or prevent altogether, the health effects of smoke exposure and environmental contaminants.”

Wildfire smoke is a toxic brew. It contains:

<ul><li>Fine particulate matter (PM2.5)</li><li>Toxic gases</li><li>Heavy metals</li><li>“Forever chemicals” like PFAS</li><li>Cancer-causing compounds</li></ul>

The ultra-fine PM2.5 particles are particularly dangerous. At less than 2.5 micrometers in diameter, they are 28 times smaller than the width of a human hair and can be inhaled deep into the lungs, enter the bloodstream, and affect organs throughout the body.

“These findings suggest that even short-term exposure to wildfire smoke can act as a biological stressor with long-lasting effects,” said Dr. Mary Johnson, lead researcher and environmental health scientist at Harvard. “That’s especially concerning as wildfire seasons grow longer and smoke plumes travel farther.”

<h2>What are the Genetic Impacts of Wildfires?</h2>

One of the most striking findings of the study was the alteration of 133 genes related to allergies and asthma in those exposed to wildfire smoke. This genetic shift may help explain why people living in wildfire-prone areas often report more respiratory problems, even months after the fires have ended.

The study also found that smoke-exposed individuals had more immune cells affected by toxic metals, further increasing inflammation and the risk of immune dysfunction. These changes may make people more susceptible to infections, worsen existing respiratory conditions, and potentially contribute to the development of new allergies or asthma.

The United States, particularly western states like California, has witnessed a dramatic increase in wildfire frequency and severity. In 2023 alone, California experienced over 7,000 wildfires, with the California Department of Forestry and Fire Protection responding to nearly 590,000 related emergencies. The devastating January 2025 fire in Los Angeles County, which claimed 30 lives and scorched 40,000 acres, underscored the deadly toll of these disasters.

Climate change is a major driver, creating hotter, drier conditions that fuel longer and more intense fire seasons. As wildfires become more common, understanding and mitigating the health risks of smoke exposure is more urgent than ever.

<h2>What are the Immediate and Long-Term Health Effects?</h2>

Wildfire smoke can trigger a range of symptoms, from burning eyes and runny noses to persistent coughs and breathing difficulties. For people with pre-existing conditions like asthma, COPD, or eczema, exposure can lead to dangerous flare-ups.

But the risks extend beyond the lungs. Fine particulate matter (PM2.5) can enter the bloodstream, causing inflammation throughout the body. Studies have linked wildfire smoke to increased emergency room visits for heart attacks and coronary heart disease within 24 hours of exposure. There is also emerging evidence that PM2.5 can impair cognitive functions such as memory and attention, likely by inducing inflammation in the brain.

<h2>How to Protect Yourself and Your Family?</h2>

Experts emphasize the need for proactive measures as wildfire season peaks:

<ul><li>Stay indoors when smoke levels are high. Keep windows closed and seal gaps in doors.</li><li>Use HEPA-grade air purifiers to reduce indoor particle pollution.</li><li>Wear a certified N95 mask when outdoors, especially if you’re in a smoke-affected region.</li><li>Monitor air quality via apps or government alerts, such as AirNow.gov.</li><li>Avoid outdoor exercise when AQI levels are poor; opt for indoor alternatives instead.</li><li>Keep emergency medications handy, especially for people with asthma or heart conditions.</li></ul>

If evacuation orders are issued, follow them promptly—not just for safety from flames, but from the health threats the smoke carries.

As wildfires continue to reshape landscapes and communities, their invisible legacy—on our immune systems and overall health—demands urgent attention. The message from scientists is clear: wildfire smoke is not just an environmental nuisance, but a profound health hazard that can affect anyone, anywhere the wind blows. Proactive measures, informed guidelines, and continued research are essential to protect public health in an increasingly fiery world.

wildfire smoke health effects

immune system and wildfire smoke

pm2 5 health risks

wildfire smoke inflammation

allergies from wildfire smoke

wildfire smoke research

wildfire safety tips

As wildfires become an all-too-familiar headline across the globe, their visible devastation—scorched forests, lost homes, and displaced communities—often overshadows a more insidious threat: the impact of wildfire smoke on human health. While the immediate dangers of flames and evacuations are clear, a new study published in Nature Medicine reveals that wildfire smoke does far more than irritate eyes and throats. It can fundamentally alter the immune system, potentially making people more susceptible to illness long after the smoke has cleared.The study, led by researchers from the Harvard T.H. Chan School of Public Health, found that people exposed to wildfire smoke exhibit changes in gene expression and immune function, even if they’re otherwise healthy. This suggests that the health consequences of wildfires may extend far beyond the immediate coughing, wheezing, and watery eyes that so many experience during fire season.How Smoke Alters Immunity?The research, led by Dr. Kari Nadeau of the Harvard T.H. Chan School of Public Health, provides the most detailed look yet at how wildfire smoke affects the body on a cellular level. By analyzing blood samples from 31 firefighters and civilians exposed to wildfire smoke and comparing them to 29 unexposed individuals, the study uncovered a complex web of immune changes.People exposed to wildfire smoke showed a marked increase in memory immune cells—cells that typically provide long-term immunity. At first glance, this might seem beneficial, but the study also found elevated biomarkers of inflammation and immune activity, as well as changes in dozens of genes linked to allergies and asthma. In short, the immune system was not just activated—it was thrown into overdrive, potentially increasing vulnerability to illness.Dr. Mary Johnson, the study’s lead researcher, explains, “Our findings demonstrate that the immune system is extremely sensitive to environmental exposures like fire smoke, even in healthy individuals. Knowing exactly how may help us detect immune dysfunction from smoke exposure earlier and could pave the way for new therapeutics to mitigate, or prevent altogether, the health effects of smoke exposure and environmental contaminants.”What’s in Wildfire Smoke? Wildfire smoke is a toxic brew. It contains:Fine particulate matter (PM2.5)Toxic gasesHeavy metals“Forever chemicals” like PFASCancer-causing compoundsThe ultra-fine PM2.5 particles are particularly dangerous. At less than 2.5 micrometers in diameter, they are 28 times smaller than the width of a human hair and can be inhaled deep into the lungs, enter the bloodstream, and affect organs throughout the body.“These findings suggest that even short-term exposure to wildfire smoke can act as a biological stressor with long-lasting effects,” said Dr. Mary Johnson, lead researcher and environmental health scientist at Harvard. “That’s especially concerning as wildfire seasons grow longer and smoke plumes travel farther.”What are the Genetic Impacts of Wildfires?One of the most striking findings of the study was the alteration of 133 genes related to allergies and asthma in those exposed to wildfire smoke. This genetic shift may help explain why people living in wildfire-prone areas often report more respiratory problems, even months after the fires have ended.The study also found that smoke-exposed individuals had more immune cells affected by toxic metals, further increasing inflammation and the risk of immune dysfunction. These changes may make people more susceptible to infections, worsen existing respiratory conditions, and potentially contribute to the development of new allergies or asthma.The United States, particularly western states like California, has witnessed a dramatic increase in wildfire frequency and severity. In 2023 alone, California experienced over 7,000 wildfires, with the California Department of Forestry and Fire Protection responding to nearly 590,000 related emergencies. The devastating January 2025 fire in Los Angeles County, which claimed 30 lives and scorched 40,000 acres, underscored the deadly toll of these disasters.Climate change is a major driver, creating hotter, drier conditions that fuel longer and more intense fire seasons. As wildfires become more common, understanding and mitigating the health risks of smoke exposure is more urgent than ever.What are the Immediate and Long-Term Health Effects?Wildfire smoke can trigger a range of symptoms, from burning eyes and runny noses to persistent coughs and breathing difficulties. For people with pre-existing conditions like asthma, COPD, or eczema, exposure can lead to dangerous flare-ups.But the risks extend beyond the lungs. Fine particulate matter (PM2.5) can enter the bloodstream, causing inflammation throughout the body. Studies have linked wildfire smoke to increased emergency room visits for heart attacks and coronary heart disease within 24 hours of exposure. There is also emerging evidence that PM2.5 can impair cognitive functions such as memory and attention, likely by inducing inflammation in the brain.How to Protect Yourself and Your Family?Experts emphasize the need for proactive measures as wildfire season peaks:Stay indoors when smoke levels are high. Keep windows closed and seal gaps in doors.Use HEPA-grade air purifiers to reduce indoor particle pollution.Wear a certified N95 mask when outdoors, especially if you’re in a smoke-affected region.Monitor air quality via apps or government alerts, such as AirNow.gov.Avoid outdoor exercise when AQI levels are poor; opt for indoor alternatives instead.Keep emergency medications handy, especially for people with asthma or heart conditions.If evacuation orders are issued, follow them promptly—not just for safety from flames, but from the health threats the smoke carries.As wildfires continue to reshape landscapes and communities, their invisible legacy—on our immune systems and overall health—demands urgent attention. The message from scientists is clear: wildfire smoke is not just an environmental nuisance, but a profound health hazard that can affect anyone, anywhere the wind blows. Proactive measures, informed guidelines, and continued research are essential to protect public health in an increasingly fiery world.

wildfire smoke may rewire your immune system, scientists warn: here’s how it impacts your health

Aarushi Bhadury

24-Year-Old Suffered A Heart Attack Due To Vaping, Now He Has Permanent Lung Damage - "I Have The Lungs Of A 70-Year-Old Now"

24-Year-Old Suffered A Heart Attack Due To Vaping, Now He Has Permanent Lung Damage

At 15, This Girl Began Walking Like A 90-Year-Old Due To A Spine Condition That Affects 1 In 10 Kids

at 15, this girl began walking like a 90-year-old due to a spine condition that affects 1 in 10 kids

Fitness

Health On Your Fingertips - Your Fingers Can Reveal How Much Endurance You Have

health on your fingertips - your fingers can reveal how much endurance you have

Sharp Elbow Pain Is Not A Good Sign For Your Heart - Cardiologist Warns Of A Little-Known Symptom

sharp elbow pain is not a good sign for your heart - cardiologist warns of a little-known symptom

Recent years have seen a rapid rise in kids vaping all over the world. A popular habit among young teens as well as young adults, vaping is the act of inhaling aerosol from electronic cigarettes or vape. But this popular habit has cost the health and well-being of this 24-year-old, who took to social media to warn people about this habit, as reported by the People. 

<h2>“I Have Been Vaping Since The Age Of 12” </h2>

A young man is taking to social media with a serious warning for anyone who uses e-cigarettes. He claims that vaping caused him to suffer a <span>heart attack</span> at just 24 years old and has led to lasting <span>lung damage</span>. Through a series of TikTok videos, Jacob Temple from Kentucky is urging people to quit vaping right away. 

Temple, who started vaping at age 12, now says his lungs are like those of a 70-year-old man. He explained that he has permanent scarring on part of his lungs, meaning he can never fully inflate or deflate them again. "It feels like I’m breathing through a straw constantly, always, just never getting enough air and there’s nothing that can be done," he shared. This damage means he'll forever feel like he's not getting enough oxygen. 

<h2>Lung Damage As Well As Suffering <span rel="noindex">Heart Health</span> </h2>

Temple also revealed that his vaping habit directly led to a "minor" heart attack, specifically an anterior myocardial infarction. He described the terrifying experience, saying, "My whole body was fighting to stay alive at a certain point." While the physical damage to his lungs can't be fixed, he hopes his story will motivate others to stop vaping before it's too late for them. 

Temple admits he still has "good days and bad days." On some days, he can be active with the help of his rescue inhaler. However, on other days, he can barely function and struggles to sleep. He reflected on his past habit, saying, "It was fun while it lasted, but now I am paying for it dearly." His struggle highlights the long-term, painful consequences of what might seem like a harmless habit. 

In a 2021 BMJ Case Reports study, researchers explored the link between vaping spontaneous pneumothorax, which is when air leaks into the space between your lung and chest wall, causing the lung to collapse. They also presented a case study of a A 34-year-old man, who was a healthy weight, came to the emergency room struggling to breathe and experiencing back pain for a full day. 

An X-ray of his chest showed a large collapsed lung on his right side. He had quit regular cigarettes three years prior and switched entirely to vaping. A more detailed CT scan revealed bullae (air-filled sacs) in his lungs, and he had to undergo surgery to remove part of his lung. Lab tests on the removed tissue showed mild inflammation. 

There are many other studies like the same, prompting researchers and healthcare professionals to issue warnings. 

Temple’s case is not an isolated incident; there are many who have suffered the same fate.

According to Heart Organization, <span>quitting smoking</span> or vaping for good can seem tough, but it's totally possible. Just take it one step at a time. Here are five easy steps to help you on your journey to a healthier life. 

<h4>Pick Your "Quit Day" and Make a Promise </h4>

Choose a day within the next week when you'll completely stop. This is your "Quit Day"! Promise yourself and tell supportive friends you're quitting. Slowly cut down before this day. 

You have options: go "cold turkey" and stop all at once, or cut down slowly each day. For example, reduce cigarettes from 20 to 10, then 5. Or take fewer puffs from each one. 

Speak with your doctor. They can tell you if medicines or other support tools, like patches or gum, would make quitting easier for you. Get their personalized advice to help you succeed.

<h4>Plan for Your Quit Day and Beyond </h4>

Get ready for your Quit Day! Have healthy snacks ready, like fruits or gum. Plan fun activities to keep busy when you feel like smoking. Try a walk, movie, or new hobby to keep your hands busy.

heart health

Heart Attack

Heart Attack Due To Vaping

Lung Damage

Lung and Heart Health

quitting smoking

Recent years have seen a rapid rise in kids vaping all over the world. A popular habit among young teens as well as young adults, vaping is the act of inhaling aerosol from electronic cigarettes or vape. But this popular habit has cost the health and well-being of this 24-year-old, who took to social media to warn people about this habit, as reported by the People. “I Have Been Vaping Since The Age Of 12” A young man is taking to social media with a serious warning for anyone who uses e-cigarettes. He claims that vaping caused him to suffer a heart attack at just 24 years old and has led to lasting lung damage. Through a series of TikTok videos, Jacob Temple from Kentucky is urging people to quit vaping right away. Temple, who started vaping at age 12, now says his lungs are like those of a 70-year-old man. He explained that he has permanent scarring on part of his lungs, meaning he can never fully inflate or deflate them again. "It feels like I’m breathing through a straw constantly, always, just never getting enough air and there’s nothing that can be done," he shared. This damage means he'll forever feel like he's not getting enough oxygen. Lung Damage As Well As Suffering Heart Health Temple also revealed that his vaping habit directly led to a "minor" heart attack, specifically an anterior myocardial infarction. He described the terrifying experience, saying, "My whole body was fighting to stay alive at a certain point." While the physical damage to his lungs can't be fixed, he hopes his story will motivate others to stop vaping before it's too late for them. Temple admits he still has "good days and bad days." On some days, he can be active with the help of his rescue inhaler. However, on other days, he can barely function and struggles to sleep. He reflected on his past habit, saying, "It was fun while it lasted, but now I am paying for it dearly." His struggle highlights the long-term, painful consequences of what might seem like a harmless habit. Research Warns Against Vaping In a 2021 BMJ Case Reports study, researchers explored the link between vaping spontaneous pneumothorax, which is when air leaks into the space between your lung and chest wall, causing the lung to collapse. They also presented a case study of a A 34-year-old man, who was a healthy weight, came to the emergency room struggling to breathe and experiencing back pain for a full day. An X-ray of his chest showed a large collapsed lung on his right side. He had quit regular cigarettes three years prior and switched entirely to vaping. A more detailed CT scan revealed bullae (air-filled sacs) in his lungs, and he had to undergo surgery to remove part of his lung. Lab tests on the removed tissue showed mild inflammation. There are many other studies like the same, prompting researchers and healthcare professionals to issue warnings. Temple’s case is not an isolated incident; there are many who have suffered the same fate. How To Quit Vaping? According to Heart Organization, quitting smoking or vaping for good can seem tough, but it's totally possible. Just take it one step at a time. Here are five easy steps to help you on your journey to a healthier life. Pick Your "Quit Day" and Make a Promise Choose a day within the next week when you'll completely stop. This is your "Quit Day"! Promise yourself and tell supportive friends you're quitting. Slowly cut down before this day. Choose How You'll Quit You have options: go "cold turkey" and stop all at once, or cut down slowly each day. For example, reduce cigarettes from 20 to 10, then 5. Or take fewer puffs from each one. Talk to Your Doctor About Help Speak with your doctor. They can tell you if medicines or other support tools, like patches or gum, would make quitting easier for you. Get their personalized advice to help you succeed. Plan for Your Quit Day and Beyond Get ready for your Quit Day! Have healthy snacks ready, like fruits or gum. Plan fun activities to keep busy when you feel like smoking. Try a walk, movie, or new hobby to keep your hands busy.

24-Year-Old Suffered A Heart Attack Due To Vaping, Now He Has Permanent Lung Damage - 'I Have The Lungs Of A 70-Year-Old Now'

24-year-old suffered a heart attack due to vaping, now he has permanent lung damage - "i have the lungs of a 70-year-old now"

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